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S Russ Price

Showing results (11-20 of 53) with videos related to

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Endocrinology|October 19, 2004
Insulin-like growth factor I: the yin and yang of muscle atrophyMaria F Chacon Heszele, S Russ Price
American Journal of Physiology. Cell Physiology|January 19, 2007
Inhibition of PI3-kinase signaling by glucocorticoids results in increased branched-chain amino acid degradation in renal epithelial cellsXiaonan Wang, Junping Hu, S Russ Price
Journal of Renal Nutrition : the Official Journal of the Council on Renal Nutrition of the National Kidney Foundation|October 2, 2022
Muscle Atrophy in CKD: A Historical Perspective of Advancements in Its UnderstandingS Russ Price, William E Mitch, Giacomo Garibotto
Nature Reviews. Nephrology|November 9, 2021
Pathophysiological mechanisms leading to muscle loss in chronic kidney diseaseXiaonan H Wang, William E Mitch, S Russ Price
Kidney International|May 20, 2004
Suppression of chaperone-mediated autophagy in the renal cortex during acute diabetes mellitusSira Sooparb, S Russ Price, Jin Shaoguang, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|October 4, 2008
Regulation of caspase-3 activity by insulin in skeletal muscle cells involves both PI3-kinase and MEK-1/2Yongmei Gao, Ronald Ordas, Janet D Klein, et al.
American Journal of Physiology. Renal Physiology|September 7, 2006
Tissue-specific regulation of ubiquitin (UbC) transcription by glucocorticoids: in vivo and in vitro analysesAnne C Marinovic, Bin Zheng, William E Mitch, et al.
The Journal of Biological Chemistry|March 2, 2002
Ubiquitin (UbC) expression in muscle cells is increased by glucocorticoids through a mechanism involving Sp1 and MEK1Anne C Marinovic, Bin Zheng, William E Mitch, et al.
Biochemical and Biophysical Research Communications|July 21, 2018
Glucocorticoid-induced CREB activation and myostatin expression in C2C12 myotubes involves phosphodiesterase-3/4 signalingYang Xie, Ben D Perry, Daniel Espinoza, et al.
Journal of the American Society of Nephrology : JASN|April 14, 2006
Chronic kidney disease causes defects in signaling through the insulin receptor substrate/phosphatidylinositol 3-kinase/Akt pathway: implications for muscle atrophyJames L Bailey, Bin Zheng, Zhaoyong Hu, et al.
Pageof 6

Showing results (11-20 of 53) with videos related to

Sort By:
Pageof 6
Endocrinology|October 19, 2004
Insulin-like growth factor I: the yin and yang of muscle atrophyMaria F Chacon Heszele, S Russ Price
American Journal of Physiology. Cell Physiology|January 19, 2007
Inhibition of PI3-kinase signaling by glucocorticoids results in increased branched-chain amino acid degradation in renal epithelial cellsXiaonan Wang, Junping Hu, S Russ Price
Journal of Renal Nutrition : the Official Journal of the Council on Renal Nutrition of the National Kidney Foundation|October 2, 2022
Muscle Atrophy in CKD: A Historical Perspective of Advancements in Its UnderstandingS Russ Price, William E Mitch, Giacomo Garibotto
Nature Reviews. Nephrology|November 9, 2021
Pathophysiological mechanisms leading to muscle loss in chronic kidney diseaseXiaonan H Wang, William E Mitch, S Russ Price
Kidney International|May 20, 2004
Suppression of chaperone-mediated autophagy in the renal cortex during acute diabetes mellitusSira Sooparb, S Russ Price, Jin Shaoguang, et al.
Journal of Applied Physiology (Bethesda, Md. : 1985)|October 4, 2008
Regulation of caspase-3 activity by insulin in skeletal muscle cells involves both PI3-kinase and MEK-1/2Yongmei Gao, Ronald Ordas, Janet D Klein, et al.
American Journal of Physiology. Renal Physiology|September 7, 2006
Tissue-specific regulation of ubiquitin (UbC) transcription by glucocorticoids: in vivo and in vitro analysesAnne C Marinovic, Bin Zheng, William E Mitch, et al.
The Journal of Biological Chemistry|March 2, 2002
Ubiquitin (UbC) expression in muscle cells is increased by glucocorticoids through a mechanism involving Sp1 and MEK1Anne C Marinovic, Bin Zheng, William E Mitch, et al.
Biochemical and Biophysical Research Communications|July 21, 2018
Glucocorticoid-induced CREB activation and myostatin expression in C2C12 myotubes involves phosphodiesterase-3/4 signalingYang Xie, Ben D Perry, Daniel Espinoza, et al.
Journal of the American Society of Nephrology : JASN|April 14, 2006
Chronic kidney disease causes defects in signaling through the insulin receptor substrate/phosphatidylinositol 3-kinase/Akt pathway: implications for muscle atrophyJames L Bailey, Bin Zheng, Zhaoyong Hu, et al.
Pageof 6