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S Schlesinger

Showing results (51-60 of 368) with videos related to

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Infection and Immunity|January 1, 1994
A role for natural antibody in the pathogenesis of leprosy: antibody in nonimmune serum mediates C3 fixation to the Mycobacterium leprae surface and hence phagocytosis by human mononuclear phagocytesL S Schlesinger, M A Horwitz
Journal of Maxillofacial Surgery|December 1, 1983
The iliac bone or osteocutaneous transplant pedicled to the deep circumflex iliac artery. II. Clinical applicationK Bitter, S Schlesinger, U Westerman
The Journal of Experimental Medicine|November 1, 1991
Phenolic glycolipid-1 of Mycobacterium leprae binds complement component C3 in serum and mediates phagocytosis by human monocytesL S Schlesinger, M A Horwitz
Diabetes Research and Clinical Practice|August 2, 2014
Impact of diabetes on the natural history of tuberculosisBlanca I Restrepo, Larry S Schlesinger
Proceedings of the National Academy of Sciences of the United States of America|July 1, 1987
Engineered defective interfering RNAs of Sindbis virus express bacterial chloramphenicol acetyltransferase in avian cellsR Levis, H Huang, S Schlesinger
Exceptional Children|February 1, 1972
Development of maturity in deaf childrenH S Schlesinger, K P Meadow
Community Mental Health Journal|February 1, 1972
A conceptual model for a program of community psychiatry for a deaf populationH S Schlesinger, K P Meadow
Journal of Virology|April 1, 1997
Sindbis virus replicons and Sindbis virus: assembly of chimeras and of particles deficient in virus RNAI Frolov, E Frolova, S Schlesinger
Journal of Immunology (Baltimore, Md. : 1950)|September 15, 1991
Phagocytosis of Mycobacterium leprae by human monocyte-derived macrophages is mediated by complement receptors CR1 (CD35), CR3 (CD11b/CD18), and CR4 (CD11c/CD18) and IFN-gamma activation inhibits complement receptor function and phagocytosis of this bacteriumL S Schlesinger, M A Horwitz
Journal of Virology|December 1, 1983
Evolution of virus and defective-interfering RNAs in BHK cells persistently infected with Sindbis virusB Weiss, R Levis, S Schlesinger
Pageof 37

Showing results (51-60 of 368) with videos related to

Sort By:
Pageof 37
Infection and Immunity|January 1, 1994
A role for natural antibody in the pathogenesis of leprosy: antibody in nonimmune serum mediates C3 fixation to the Mycobacterium leprae surface and hence phagocytosis by human mononuclear phagocytesL S Schlesinger, M A Horwitz
Journal of Maxillofacial Surgery|December 1, 1983
The iliac bone or osteocutaneous transplant pedicled to the deep circumflex iliac artery. II. Clinical applicationK Bitter, S Schlesinger, U Westerman
The Journal of Experimental Medicine|November 1, 1991
Phenolic glycolipid-1 of Mycobacterium leprae binds complement component C3 in serum and mediates phagocytosis by human monocytesL S Schlesinger, M A Horwitz
Diabetes Research and Clinical Practice|August 2, 2014
Impact of diabetes on the natural history of tuberculosisBlanca I Restrepo, Larry S Schlesinger
Proceedings of the National Academy of Sciences of the United States of America|July 1, 1987
Engineered defective interfering RNAs of Sindbis virus express bacterial chloramphenicol acetyltransferase in avian cellsR Levis, H Huang, S Schlesinger
Exceptional Children|February 1, 1972
Development of maturity in deaf childrenH S Schlesinger, K P Meadow
Community Mental Health Journal|February 1, 1972
A conceptual model for a program of community psychiatry for a deaf populationH S Schlesinger, K P Meadow
Journal of Virology|April 1, 1997
Sindbis virus replicons and Sindbis virus: assembly of chimeras and of particles deficient in virus RNAI Frolov, E Frolova, S Schlesinger
Journal of Immunology (Baltimore, Md. : 1950)|September 15, 1991
Phagocytosis of Mycobacterium leprae by human monocyte-derived macrophages is mediated by complement receptors CR1 (CD35), CR3 (CD11b/CD18), and CR4 (CD11c/CD18) and IFN-gamma activation inhibits complement receptor function and phagocytosis of this bacteriumL S Schlesinger, M A Horwitz
Journal of Virology|December 1, 1983
Evolution of virus and defective-interfering RNAs in BHK cells persistently infected with Sindbis virusB Weiss, R Levis, S Schlesinger
Pageof 37