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British Journal of Haematology|June 1, 1984
(A gamma delta beta) thalassaemia: similarity of phenotype in four different molecular defects, including one newly describedR J Trent, R W Jones, J B Clegg, et al.British Journal of Haematology|February 26, 2000
Rapid detection of alpha-thalassaemia deletions and alpha-globin gene triplication by multiplex polymerase chain reactionsY T Liu, J M Old, K Miles, et al.Nature|November 24, 1983
Alpha-thalassaemia caused by a polyadenylation signal mutationD R Higgs, S E Goodbourn, J Lamb, et al.Blood|February 1, 1988
The polyadenylation site mutation in the alpha-globin gene clusterS L Thein, R B Wallace, L Pressley, et al.Leukemia Research|January 1, 1984
Erythroid differentiation in CGL cells from a patient with blast crisisC G Potter, C Bunch, A C Potter, et al.The Journal of Clinical Investigation|January 1, 1987
Different hematologic phenotypes are associated with the leftward (-alpha 4.2) and rightward (-alpha 3.7) alpha+-thalassemia deletionsD K Bowden, A V Hill, D R Higgs, et al.Molecular Biology & Medicine|July 1, 1983
Globin gene mapping studies in Sardinian patients homozygous for beta zero ThalassaemiaJ S Wainscoat, J I Bell, J M Old, et al.Lancet (London, England)|July 2, 1983
Aplastic crisis due to parvovirus infection in pyruvate kinase deficiencyJ R Duncan, C B Potter, M D Cappellini, et al.Progress in Clinical and Biological Research|January 1, 1987
Raised Hb F levels in sickle cell disease are caused by a determinant linked to the beta globin gene clusterA E Kulozik, S L Thein, B C Kar, et al.Blood|June 1, 1987
Fetal hemoglobin levels and beta (s) globin haplotypes in an Indian populations with sickle cell diseaseA E Kulozik, B C Kar, R K Satapathy, et al.Pageof 28