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S Welch

Showing results (381-390 of 422) with videos related to

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Nature|March 5, 2014
Protection against filovirus diseases by a novel broad-spectrum nucleoside analogue BCX4430Travis K Warren, Jay Wells, Rekha G Panchal, et al.
Haematologica|March 3, 2022
Decitabine salvage for <i>TP53</i>-mutated, relapsed/refractory acute myeloid leukemia after cytotoxic induction therapyFrancesca Ferraro, Agata Gruszczynska, Marianna B Ruzinova, et al.
Nature Medicine|October 20, 2014
Age-related mutations associated with clonal hematopoietic expansion and malignanciesMingchao Xie, Charles Lu, Jiayin Wang, et al.
Cell|February 21, 2017
CpG Island Hypermethylation Mediated by DNMT3A Is a Consequence of AML ProgressionDavid H Spencer, David A Russler-Germain, Shamika Ketkar, et al.
JAMA|April 21, 2011
Use of whole-genome sequencing to diagnose a cryptic fusion oncogeneJohn S Welch, Peter Westervelt, Li Ding, et al.
Blood|January 14, 2017
Mutational landscape and response are conserved in peripheral blood of AML and MDS patients during decitabine therapyEric J Duncavage, Geoffrey L Uy, Allegra A Petti, et al.
Nature Communications|February 2, 2018
Cellular stressors contribute to the expansion of hematopoietic clones of varying leukemic potentialTerrence N Wong, Christopher A Miller, Matthew R M Jotte, et al.
Nature|December 10, 2014
Role of TP53 mutations in the origin and evolution of therapy-related acute myeloid leukaemiaTerrence N Wong, Giridharan Ramsingh, Andrew L Young, et al.
Leukemia|October 15, 2016
Dynamic changes in the clonal structure of MDS and AML in response to epigenetic therapyG L Uy, E J Duncavage, G S Chang, et al.
Current Oncology (Toronto, Ont.)|July 24, 2010
Eastern Canadian Colorectal Cancer Consensus Conference: setting the limits of resectable diseaseM Vickers, B Samson, B Colwell, et al.
Pageof 43

Showing results (381-390 of 422) with videos related to

Sort By:
Pageof 43
Nature|March 5, 2014
Protection against filovirus diseases by a novel broad-spectrum nucleoside analogue BCX4430Travis K Warren, Jay Wells, Rekha G Panchal, et al.
Haematologica|March 3, 2022
Decitabine salvage for <i>TP53</i>-mutated, relapsed/refractory acute myeloid leukemia after cytotoxic induction therapyFrancesca Ferraro, Agata Gruszczynska, Marianna B Ruzinova, et al.
Nature Medicine|October 20, 2014
Age-related mutations associated with clonal hematopoietic expansion and malignanciesMingchao Xie, Charles Lu, Jiayin Wang, et al.
Cell|February 21, 2017
CpG Island Hypermethylation Mediated by DNMT3A Is a Consequence of AML ProgressionDavid H Spencer, David A Russler-Germain, Shamika Ketkar, et al.
JAMA|April 21, 2011
Use of whole-genome sequencing to diagnose a cryptic fusion oncogeneJohn S Welch, Peter Westervelt, Li Ding, et al.
Blood|January 14, 2017
Mutational landscape and response are conserved in peripheral blood of AML and MDS patients during decitabine therapyEric J Duncavage, Geoffrey L Uy, Allegra A Petti, et al.
Nature Communications|February 2, 2018
Cellular stressors contribute to the expansion of hematopoietic clones of varying leukemic potentialTerrence N Wong, Christopher A Miller, Matthew R M Jotte, et al.
Nature|December 10, 2014
Role of TP53 mutations in the origin and evolution of therapy-related acute myeloid leukaemiaTerrence N Wong, Giridharan Ramsingh, Andrew L Young, et al.
Leukemia|October 15, 2016
Dynamic changes in the clonal structure of MDS and AML in response to epigenetic therapyG L Uy, E J Duncavage, G S Chang, et al.
Current Oncology (Toronto, Ont.)|July 24, 2010
Eastern Canadian Colorectal Cancer Consensus Conference: setting the limits of resectable diseaseM Vickers, B Samson, B Colwell, et al.
Pageof 43