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Protein Engineering, Design & Selection : PEDS|September 1, 2018
Synthetic 10FN3-based mono- and bivalent inhibitors of MDM2/X functionS-Y Lau, J W Siau, R M Sobota, et al.Neuropharmacology|December 15, 2010
Isolation and identification of anti-inflammatory constituents from Ligusticum chuanxiong and their underlying mechanisms of action on microgliaTerry C T Or, Cindy L H Yang, Anna H Y Law, et al.Journal of Leukocyte Biology|April 30, 2009
A role for glycogen synthase kinase-3 in antagonizing mycobacterial immune evasion by negatively regulating IL-10 inductionMabel M P Chan, Benny K W Cheung, James C B Li, et al.Journal of Virology|October 13, 2006
Role for nonstructural protein 1 of severe acute respiratory syndrome coronavirus in chemokine dysregulationAnna H Y Law, Davy C W Lee, Benny K W Cheung, et al.Chemico-Biological Interactions|December 8, 2009
Senkyunolides reduce hydrogen peroxide-induced oxidative damage in human liver HepG2 cells via induction of heme oxygenase-1Hongyi Qi, Shiu On Siu, Yan Chen, et al.Journal of Medicinal Chemistry|October 20, 2009
Identification of the bioactive constituent and its mechanisms of action in mediating the anti-inflammatory effects of black cohosh and related Cimicifuga species on human primary blood macrophagesCindy L H Yang, Stanley C C Chik, James C B Li, et al.Scientific Reports|March 18, 2022
Post-lockdown changes of age-specific susceptibility and its correlation with adherence to social distancing measuresMax S Y Lau, Carol Liu, Aaron J Siegler, et al.Journal of Virology|July 30, 2005
p38 mitogen-activated protein kinase-dependent hyperinduction of tumor necrosis factor alpha expression in response to avian influenza virus H5N1Davy C W Lee, Chung-Yan Cheung, Anna H Y Law, et al.American Journal of Perinatology|August 25, 2012
Bacillus bloodstream infections in a tertiary perinatal centre: an 8-year studyJoseph Yuk Ting, Samson Sai-yin Wong, Yuet-yee Chee, et al.Scientific Reports|July 30, 2016
Trophoblastic debris modifies endothelial cell transcriptome in vitro: a mechanism by which fetal cells might control maternal responses to pregnancyJ Wei, S Y Lau, C Blenkiron, et al.Pageof 15