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Cell Reports|September 30, 2016
Mesenchymal Transition of High-Grade Breast Carcinomas Depends on Extracellular Matrix Control of Myeloid Suppressor Cell ActivitySabina Sangaletti, Claudio Tripodo, Alessandra Santangelo, et al.
Cancer Discovery|November 6, 2013
Defective stromal remodeling and neutrophil extracellular traps in lymphoid tissues favor the transition from autoimmunity to lymphomaSabina Sangaletti, Claudio Tripodo, Caterina Vitali, et al.
Cancer Research|November 29, 2019
Transcriptional Profiles and Stromal Changes Reveal Bone Marrow Adaptation to Early Breast Cancer in Association with Deregulated Circulating microRNAsClaudia Chiodoni, Valeria Cancila, Tiziana A Renzi, et al.
Frontiers in Immunology|July 9, 2019
SPARC Is a New Myeloid-Derived Suppressor Cell Marker Licensing Suppressive ActivitiesSabina Sangaletti, Giovanna Talarico, Claudia Chiodoni, et al.
Angiogenesis|December 5, 2024
Extracellular nicotinamide phosphoribosyltransferase (eNAMPT) drives abnormal pericyte-rich vasculature in triple-negative breast cancerMarianna Moro, Federica Carolina Balestrero, Giorgia Colombo, et al.
Cancer Cell|August 13, 2015
RORC1 Regulates Tumor-Promoting "Emergency" Granulo-MonocytopoiesisLaura Strauss, Sabina Sangaletti, Francesca Maria Consonni, et al.
Journal of Experimental & Clinical Cancer Research : CR|June 16, 2026
ZEB1 integrates stemness, differentiation blockade and immune suppression to orchestrate osteosarcoma aggressivenessAnna Zaccaria, Caterina Cascini, Chiara Ratti, et al.
Frontiers in Immunology|June 4, 2021
CD40 Activity on Mesenchymal Cells Negatively Regulates OX40L to Maintain Bone Marrow Immune Homeostasis Under Stress ConditionsBarbara Bassani, Claudio Tripodo, Paola Portararo, et al.
Clinical Lung Cancer|August 27, 2024
The EXcellenT Trial: Exercise in Extended Oncogene Addicted Lung Cancer in Active TreatmentChiara Bennati, Roberto Ferrara, Sabina Sangaletti, et al.
Journal of Autoimmunity|October 4, 2011
The matricellular protein SPARC supports follicular dendritic cell networking toward Th17 responsesSilvia Piconese, Massimo Costanza, Claudio Tripodo, et al.
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