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The Journal of Clinical Investigation|July 26, 2011
Oxidized CaMKII: a "heart stopper" for the sinus node?Sabine Huke, Björn C KnollmannJournal of Molecular and Cellular Cardiology|January 26, 2010
Increased myofilament Ca2+-sensitivity and arrhythmia susceptibilitySabine Huke, Björn C KnollmannCirculation Research|March 28, 2013
Focal energy deprivation underlies arrhythmia susceptibility in mice with calcium-sensitized myofilamentsSabine Huke, Raghav Venkataraman, Michela Faggioni, et al.Circulation Research|June 1, 2012
Myofilament Ca sensitization increases cytosolic Ca binding affinity, alters intracellular Ca homeostasis, and causes pause-dependent Ca-triggered arrhythmiaTilmann Schober, Sabine Huke, Raghav Venkataraman, et al.The Journal of Physiology|May 20, 2009
New roles of calsequestrin and triadin in cardiac muscleBjörn C KnollmannCirculation Research|April 10, 2013
Induced pluripotent stem cell-derived cardiomyocytes: boutique science or valuable arrhythmia model?Björn C KnollmannThe Journal of Physiology|February 17, 2017
Linking myofilaments to sudden cardiac death: recent advancesSabine HukeJournal of Molecular and Cellular Cardiology|October 20, 2009
Flecainide inhibits arrhythmogenic Ca2+ waves by open state block of ryanodine receptor Ca2+ release channels and reduction of Ca2+ spark massFredrick A Hilliard, Derek S Steele, Derek Laver, et al.Archives of Biochemistry and Biophysics|November 17, 2018
Pathogenic troponin T mutants with opposing effects on myofilament Ca<sup>2+</sup> sensitivity attenuate cardiomyopathy phenotypes in miceKarissa M Dieseldorff Jones, Yeojung Koh, Rebecca S Weller, et al.Journal of Cardiovascular Electrophysiology|July 1, 2009
Cardiac calsequestrin: the new kid on the block in arrhythmiasNagesh Chopra, Björn C KnollmannPageof 11