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Journal of Cardiovascular Development and Disease|July 25, 2022
Left Ventricular Noncompaction in Children: The Role of Genetics, Morphology, and Function for OutcomeSabine Klaassen, Jirko Kühnisch, Alina Schultze-Berndt, et al.European Radiology|July 10, 2012
Value of cardiovascular MR in diagnosing left ventricular non-compaction cardiomyopathy and in discriminating between other cardiomyopathiesMatthias Grothoff, Milena Pachowsky, Janine Hoffmann, et al.Plos One|January 9, 2014
Outlier-based identification of copy number variations using targeted resequencing in a small cohort of patients with Tetralogy of FallotVikas Bansal, Cornelia Dorn, Marcel Grunert, et al.Frontiers in Cardiovascular Medicine|March 7, 2022
Midwall Fibrosis and Cardiac Mechanics: Rigid Body Rotation Is a Novel Marker of Disease Severity in Pediatric Primary Dilated CardiomyopathyNadya Al-Wakeel-Marquard, Franziska Seidel, Jirko Kühnisch, et al.International Journal of Cardiology|March 19, 2021
Diffuse myocardial fibrosis by T1 mapping is associated with heart failure in pediatric primary dilated cardiomyopathyNadya Al-Wakeel-Marquard, Franziska Seidel, Christopher Herbst, et al.The American Journal of Cardiology|November 1, 2011
Predictors of adverse outcome in adolescents and adults with isolated left ventricular noncompactionMatthias Greutmann, May Ling Mah, Candice K Silversides, et al.Journal of the American Heart Association|February 18, 2022
Nationwide Registry-Based Analysis of Infective Endocarditis Risk After Pulmonary Valve ReplacementClara Stammnitz, Dörte Huscher, Ulrike M M Bauer, et al.Circulation. Cardiovascular Genetics|May 10, 2011
Sarcomere gene mutations in isolated left ventricular noncompaction cardiomyopathy do not predict clinical phenotypeSusanne Probst, Erwin Oechslin, Pia Schuler, et al.HGG Advances|April 1, 2026
A homozygous variant in cardiac troponin I3, TNNI3, causes severe pediatric restrictive cardiomyopathyJirko Kühnisch, Cara L Barnett, Josephine Brendel, et al.Human Molecular Genetics|July 29, 2016
The progressive ankylosis protein ANK facilitates clathrin- and adaptor-mediated membrane traffic at the trans-Golgi network-to-endosome interfaceWenke Seifert, York Posor, Peter Schu, et al.Pageof 5