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Life Science Alliance|December 14, 2020
Requirement of DNMT1 to orchestrate epigenomic reprogramming for NPM-ALK-driven lymphomagenesisElisa Redl, Raheleh Sheibani-Tezerji, Crhistian de Jesus Cardona, et al.Molecular Cancer|March 31, 2022
KMT2C methyltransferase domain regulated INK4A expression suppresses prostate cancer metastasisTanja Limberger, Michaela Schlederer, Karolina Trachtová, et al.The EMBO Journal|September 29, 2021
DNA hypomethylation leads to cGAS-induced autoinflammation in the epidermisMirjam A Beck, Heinz Fischer, Lisa M Grabner, et al.Molecular Systems Biology|April 24, 2020
STAT3-dependent analysis reveals PDK4 as independent predictor of recurrence in prostate cancerMonika Oberhuber, Matteo Pecoraro, Mate Rusz, et al.Molecular Cancer|November 1, 2024
Cell-autonomous IL6ST activation suppresses prostate cancer development via STAT3/ARF/p53-driven senescence and confers an immune-active tumor microenvironmentChristina Sternberg, Martin Raigel, Tanja Limberger, et al.Molecular Cancer|May 29, 2024
JUN mediates the senescence associated secretory phenotype and immune cell recruitment to prevent prostate cancer progressionTorben Redmer, Martin Raigel, Christina Sternberg, et al.Nature Medicine|October 16, 2012
PDGFR blockade is a rational and effective therapy for NPM-ALK-driven lymphomasDaniela Laimer, Helmut Dolznig, Karoline Kollmann, et al.Molecular Cancer|August 12, 2023
STAT3/LKB1 controls metastatic prostate cancer by regulating mTORC1/CREB pathwayJan Pencik, Cecile Philippe, Michaela Schlederer, et al.Pageof 3