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Molecular Therapy : the Journal of the American Society of Gene Therapy|September 5, 2013
Cardiac progenitor cells engineered with βARKct have enhanced β-adrenergic toleranceMohsin Khan, Sadia Mohsin, Haruhiro Toko, et al.
Circulation Research|June 9, 2017
Role of STIM1 (Stromal Interaction Molecule 1) in Hypertrophy-Related Contractile DysfunctionConstantine D Troupes, Markus Wallner, Giulia Borghetti, et al.
Clinical and Translational Science|June 17, 2014
Imatinib activates pathological hypertrophy by altering myocyte calcium regulationLarry A Barr, Catherine A Makarewich, Remus M Berretta, et al.
Circulation Research|October 8, 2005
Ca2+ influx-induced sarcoplasmic reticulum Ca2+ overload causes mitochondrial-dependent apoptosis in ventricular myocytesXiongwen Chen, Xiaoying Zhang, Hajime Kubo, et al.
Journal of Molecular and Cellular Cardiology|September 1, 2009
Pleiotropic effects of neutrophils on myocyte apoptosis and left ventricular remodeling during early volume overloadMikhail A Kolpakov, Rachid Seqqat, Khadija Rafiq, et al.
Journal of Molecular and Cellular Cardiology|August 28, 2012
Ca(2+) influx through L-type Ca(2+) channels and transient receptor potential channels activates pathological hypertrophy signalingHui Gao, Fang Wang, Wei Wang, et al.
Molecular Vision|February 18, 2010
A new locus for autosomal recessive congenital cataract identified in a Pakistani familyHaiba Kaul, S Amer Riazuddin, Afshan Yasmeen, et al.
Frontiers in Medicine|June 30, 2017
microRNA in Cardiovascular Aging and Age-Related Cardiovascular DiseasesClaudio de Lucia, Klara Komici, Giulia Borghetti, et al.
Circulation Research|October 22, 2014
GRK5-mediated exacerbation of pathological cardiac hypertrophy involves facilitation of nuclear NFAT activityJonathan E Hullmann, Laurel A Grisanti, Catherine A Makarewich, et al.
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