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Journal of Molecular and Cellular Cardiology|June 8, 2013
T-type Ca²⁺ channels regulate the exit of cardiac myocytes from the cell cycle after birthFang Wang, Hui Gao, Hajime Kubo, et al.Circulation Research|June 27, 2013
Bone-derived stem cells repair the heart after myocardial infarction through transdifferentiation and paracrine signaling mechanismsJason M Duran, Catherine A Makarewich, Thomas E Sharp, et al.The Journal of Physiology|December 24, 2017
Increasing T-type calcium channel activity by β-adrenergic stimulation contributes to β-adrenergic regulation of heart ratesYingxin Li, Xiaoxiao Zhang, Chen Zhang, et al.Circulation Research|December 18, 2012
β-Adrenergic regulation of cardiac progenitor cell death versus survival and proliferationMohsin Khan, Sadia Mohsin, Daniele Avitabile, et al.Redox Biology|May 2, 2024
SAH is a major metabolic sensor mediating worsening metabolic crosstalk in metabolic syndromeRamon Cueto, Wen Shen, Lu Liu, et al.Development (Cambridge, England)|October 29, 2024
A neuro-lymphatic communication guides lymphatic development by CXCL12 and CXCR4 signalingLong Nguyen Hoang Do, Esteban Delgado, Casey G Lim, et al.Journal of the American College of Cardiology|January 17, 2015
Nucleostemin rejuvenates cardiac progenitor cells and antagonizes myocardial agingNirmala Hariharan, Pearl Quijada, Sadia Mohsin, et al.Redox Biology|October 10, 2021
LIN28a induced metabolic and redox regulation promotes cardiac cell survival in the heart after ischemic injuryAntonia Elizabeth Yuko, Vagner Oliveira Carvalho Rigaud, Justin Kurian, et al.Circulation Research|April 28, 2017
Concurrent Isolation of 3 Distinct Cardiac Stem Cell Populations From a Single Human Heart BiopsyMegan M Monsanto, Kevin S White, Taeyong Kim, et al.American Journal of Physiology. Heart and Circulatory Physiology|August 4, 2023
Effects of maternal hypothyroidism on postnatal cardiomyocyte proliferation and cardiac disease responses of the progenyYijia Li, Jaslyn P Johnson, Yijun Yang, et al.Pageof 24