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Proceedings of the National Academy of Sciences of the United States of America|July 16, 2008
Preprocalcitonin signal peptide generates a cytotoxic T lymphocyte-defined tumor epitope processed by a proteasome-independent pathwayFaten El Hage, Vincent Stroobant, Isabelle Vergnon, et al.
Critical Reviews in Immunology|April 20, 2019
Tumor Hypoxia: A Key Determinant of Microenvironment Hostility and a Major Checkpoint during the Antitumor ResponseAmirtharaj Francis, Goutham Hassan Venkatesh, Rania Faouzi Zaarour, et al.
Bulletin Du Cancer|November 12, 2003
[Tumor/cytotoxic effector cross-talk in the control of tumor susceptibility to lysis]Asma Gati, Guillaume Dorothée, Jérôme Thiéry, et al.
Blood|March 6, 2002
Leukemic target susceptibility to natural killer cytotoxicity: relationship with BCR-ABL expressionFrédéric Baron, Ali G Turhan, Julien Giron-Michel, et al.
Journal of Immunology (Baltimore, Md. : 1950)|January 6, 2006
The decreased susceptibility of Bcr/Abl targets to NK cell-mediated lysis in response to imatinib mesylate involves modulation of NKG2D ligands, GM1 expression, and synapse formationChristelle Cebo, Sylvie Da Rocha, Sebastian Wittnebel, et al.
International Immunology|April 29, 2008
Potentiation of NK cell-mediated cytotoxicity in human lung adenocarcinoma: role of NKG2D-dependent pathwayBéatrice Le Maux Chansac, Dorothée Missé, Catherine Richon, et al.
Cancer Research|September 12, 2015
Fas Ligand Deficiency Impairs Tumor Immunity by Promoting an Accumulation of Monocytic Myeloid-Derived Suppressor CellsSanam Peyvandi, Stéphanie Buart, Boubekeur Samah, et al.
Oncoimmunology|November 21, 2015
miR-181a modulates acute myeloid leukemia susceptibility to natural killer cellsArash Nanbakhsh, Géraldine Visentin, Daniel Olive, et al.
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