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Plos One|August 14, 2008
Impaired adult neurogenesis in the dentate gyrus of a triple transgenic mouse model of Alzheimer's diseaseJosé J Rodríguez, Victoria C Jones, Masashi Tabuchi, et al.
Aging Cell|January 4, 2012
Lifelong rapamycin administration ameliorates age-dependent cognitive deficits by reducing IL-1β and enhancing NMDA signalingSmita Majumder, Antonella Caccamo, David X Medina, et al.
Life Sciences|May 15, 2012
Sortilin is required for toxic action of Aβ oligomers (AβOs): extracellular AβOs trigger apoptosis, and intraneuronal AβOs impair degradation pathwaysAyumi Takamura, Yota Sato, Daisuke Watabe, et al.
Molecular Psychiatry|January 10, 2019
Maternal choline supplementation ameliorates Alzheimer's disease pathology by reducing brain homocysteine levels across multiple generationsRamon Velazquez, Eric Ferreira, Wendy Winslow, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 16, 2015
Reducing Ribosomal Protein S6 Kinase 1 Expression Improves Spatial Memory and Synaptic Plasticity in a Mouse Model of Alzheimer's DiseaseAntonella Caccamo, Caterina Branca, Joshua S Talboom, et al.
Behavioural Brain Research|October 4, 2008
Validation of a 2-day water maze protocol in miceMaria Gulinello, Michael Gertner, Guadalupe Mendoza, et al.
Neuron|August 5, 2003
Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunctionSalvatore Oddo, Antonella Caccamo, Jason D Shepherd, et al.
The Journal of Biological Chemistry|February 23, 2013
β2 adrenergic receptor, protein kinase A (PKA) and c-Jun N-terminal kinase (JNK) signaling pathways mediate tau pathology in Alzheimer disease modelsDayong Wang, Qin Fu, Yuan Zhou, et al.
The Journal of Clinical Investigation|July 3, 2004
Caspase-cleavage of tau is an early event in Alzheimer disease tangle pathologyRobert A Rissman, Wayne W Poon, Mathew Blurton-Jones, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 25, 2002
Inclusion body myositis-like phenotype induced by transgenic overexpression of beta APP in skeletal muscleMichael C Sugarman, Tritia R Yamasaki, Salvatore Oddo, et al.
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