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Frontiers in Neurology|June 10, 2021
Exercise Factors Released by the Liver, Muscle, and Bones Have Promising Therapeutic Potential for StrokeJoseph S Stephan, Sama F Sleiman
Current Opinion in Neurology|October 1, 2019
Exercise factors as potential mediators of cognitive rehabilitation following traumatic brain injuryJoseph S Stephan, Sama F Sleiman
Brain Plasticity (Amsterdam, Netherlands)|May 17, 2018
Downstream Consequences of Exercise Through the Action of BDNFSama F Sleiman, Moses V Chao
Mechanisms of Development|September 3, 2011
Developmental functions for the Caenorhabditis elegans Sp protein SPTF-3Elizabeth A Ulm, Sama F Sleiman, Helen M Chamberlin
Genetics|August 28, 2007
Modulation of Caenorhabditis elegans transcription factor activity by HIM-8 and the related Zinc-Finger ZIM proteinsHongliu Sun, Brian L Nelms, Sama F Sleiman, et al.
Mechanisms of Development|June 1, 2005
Alteration of the DNA binding domain disrupts distinct functions of the C. elegans Pax protein EGL-38Guojuan Zhang, Sama F Sleiman, Rong-Jeng Tseng, et al.
Pharmaceuticals (Basel, Switzerland)|May 15, 2012
Histone Deacetylase Inhibitors and Mithramycin A Impact a Similar Neuroprotective Pathway at a Crossroad between Cancer and NeurodegenerationSama F Sleiman, Jill Berlin, Manuela Basso, et al.
Expert Opinion on Investigational Drugs|April 25, 2009
Putting the 'HAT' back on survival signalling: the promises and challenges of HDAC inhibition in the treatment of neurological conditionsSama F Sleiman, Manuela Basso, Lata Mahishi, et al.
Journal of Neuroscience Research|March 5, 2013
In vitro ischemia suppresses hypoxic induction of hypoxia-inducible factor-1α by inhibition of synthesis and not enhanced degradationSaravanan S Karuppagounder, Manuela Basso, Sama F Sleiman, et al.
Chemistry & Biology|April 21, 2015
Hydroxamate-based histone deacetylase inhibitors can protect neurons from oxidative stress via a histone deacetylase-independent catalase-like mechanismDavid E Olson, Sama F Sleiman, Megan W Bourassa, et al.
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