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Samantha J Clarke

Showing results (1-10 of 9) with videos related to

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Biochimica Et Biophysica Acta|July 17, 2007
The role of mitochondria in protection of the heart by preconditioningAndrew P Halestrap, Samantha J Clarke, Igor Khaliulin
The Journal of Biological Chemistry|July 4, 2002
Sanglifehrin A acts as a potent inhibitor of the mitochondrial permeability transition and reperfusion injury of the heart by binding to cyclophilin-D at a different site from cyclosporin ASamantha J Clarke, Gavin P McStay, Andrew P Halestrap
The Biochemical Journal|August 1, 2002
Role of critical thiol groups on the matrix surface of the adenine nucleotide translocase in the mechanism of the mitochondrial permeability transition poreGavin P McStay, Samantha J Clarke, Andrew P Halestrap
Cardiovascular Research|February 14, 2004
Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotectionAndrew P Halestrap, Samantha J Clarke, Sabzali A Javadov
Biochimie|May 23, 2002
The permeability transition pore complex: another viewAndrew P Halestrap, Gavin P McStay, Samantha J Clarke
The Journal of Physiology|March 31, 2007
Temperature preconditioning of isolated rat hearts--a potent cardioprotective mechanism involving a reduction in oxidative stress and inhibition of the mitochondrial permeability transition poreIgor Khaliulin, Samantha J Clarke, Hua Lin, et al.
Circulation Research|March 22, 2008
Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylationSamantha J Clarke, Igor Khaliulin, Manika Das, et al.
The Journal of Physiology|December 17, 2002
The effects of ischaemic preconditioning, diazoxide and 5-hydroxydecanoate on rat heart mitochondrial volume and respirationKelvin H H Lim, Sabzali A Javadov, Manika Das, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 8, 2007
Urocortin prevents mitochondrial permeability transition in response to reperfusion injury indirectly by reducing oxidative stressPaul A Townsend, Sean M Davidson, Samantha J Clarke, et al.
Pageof 1

Showing results (1-10 of 9) with videos related to

Sort By:
Pageof 1
Biochimica Et Biophysica Acta|July 17, 2007
The role of mitochondria in protection of the heart by preconditioningAndrew P Halestrap, Samantha J Clarke, Igor Khaliulin
The Journal of Biological Chemistry|July 4, 2002
Sanglifehrin A acts as a potent inhibitor of the mitochondrial permeability transition and reperfusion injury of the heart by binding to cyclophilin-D at a different site from cyclosporin ASamantha J Clarke, Gavin P McStay, Andrew P Halestrap
The Biochemical Journal|August 1, 2002
Role of critical thiol groups on the matrix surface of the adenine nucleotide translocase in the mechanism of the mitochondrial permeability transition poreGavin P McStay, Samantha J Clarke, Andrew P Halestrap
Cardiovascular Research|February 14, 2004
Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotectionAndrew P Halestrap, Samantha J Clarke, Sabzali A Javadov
Biochimie|May 23, 2002
The permeability transition pore complex: another viewAndrew P Halestrap, Gavin P McStay, Samantha J Clarke
The Journal of Physiology|March 31, 2007
Temperature preconditioning of isolated rat hearts--a potent cardioprotective mechanism involving a reduction in oxidative stress and inhibition of the mitochondrial permeability transition poreIgor Khaliulin, Samantha J Clarke, Hua Lin, et al.
Circulation Research|March 22, 2008
Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylationSamantha J Clarke, Igor Khaliulin, Manika Das, et al.
The Journal of Physiology|December 17, 2002
The effects of ischaemic preconditioning, diazoxide and 5-hydroxydecanoate on rat heart mitochondrial volume and respirationKelvin H H Lim, Sabzali A Javadov, Manika Das, et al.
American Journal of Physiology. Heart and Circulatory Physiology|May 8, 2007
Urocortin prevents mitochondrial permeability transition in response to reperfusion injury indirectly by reducing oxidative stressPaul A Townsend, Sean M Davidson, Samantha J Clarke, et al.
Pageof 1