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Frontiers in Immunology|November 22, 2021
SARS-CoV-2 Exacerbates COVID-19 Pathology Through Activation of the Complement and Kinin SystemsAnne G Savitt, Samantha Manimala, Tiara White, et al.Elife|April 1, 2020
Drosophila TRIM32 cooperates with glycolytic enzymes to promote cell growthSimranjot Bawa, David S Brooks, Kathryn E Neville, et al.Proceedings of the National Academy of Sciences of the United States of America|September 30, 2010
Allosteric inhibition of complement function by a staphylococcal immune evasion proteinHui Chen, Daniel Ricklin, Michal Hammel, et al.Journal of Immunology (Baltimore, Md. : 1950)|July 24, 2009
A molecular insight into complement evasion by the staphylococcal complement inhibitor protein familyDaniel Ricklin, Apostolia Tzekou, Brandon L Garcia, et al.Protein Science : a Publication of the Protein Society|November 9, 2017
Evidence for multiple modes of neutrophil serine protease recognition by the EAP family of Staphylococcal innate immune evasion proteinsDaphne A C Stapels, Jordan L Woehl, Fin J Milder, et al.Journal of Immunology (Baltimore, Md. : 1950)|December 27, 2023
Inhibition of the C1s Protease and the Classical Complement Pathway by 6-(4-Phenylpiperazin-1-yl)Pyridine-3-Carboximidamide and Chemical AnalogsXin Xu, Timothy J Herdendorf, Huiquan Duan, et al.The Journal of Biological Chemistry|February 8, 2018
Identification of a staphylococcal complement inhibitor with broad host specificity in equid Staphylococcus aureus strainsNienke W M de Jong, Manouk Vrieling, Brandon L Garcia, et al.Proceedings of the National Academy of Sciences of the United States of America|August 28, 2014
Staphylococcus aureus secretes a unique class of neutrophil serine protease inhibitorsDaphne A C Stapels, Kasra X Ramyar, Markus Bischoff, et al.Science (New York, N.Y.)|January 26, 2008
NFAT binding and regulation of T cell activation by the cytoplasmic scaffolding Homer proteinsGuo N Huang, David L Huso, Samuel Bouyain, et al.Journal of Immunology (Baltimore, Md. : 1950)|November 9, 2014
The extracellular adherence protein from Staphylococcus aureus inhibits the classical and lectin pathways of complement by blocking formation of the C3 proconvertaseJordan L Woehl, Daphne A C Stapels, Brandon L Garcia, et al.Pageof 14