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The Journal of Experimental Medicine|February 5, 2003
Activation of dendritic cells through the interleukin 1 receptor 1 is critical for the induction of autoimmune myocarditisUrs Eriksson, Michael O Kurrer, Ivo Sonderegger, et al.
The Journal of Cell Biology|March 24, 2010
Fibroblast growth factor receptors 1 and 2 in keratinocytes control the epidermal barrier and cutaneous homeostasisJingxuan Yang, Michael Meyer, Anna-Katharina Müller, et al.
European Journal of Immunology|June 26, 2012
Cooperation of Th1 and Th17 cells determines transition from autoimmune myocarditis to dilated cardiomyopathyVeronika Nindl, Reinhard Maier, David Ratering, et al.
The Journal of Experimental Medicine|September 24, 2020
The GM-CSF-IRF5 signaling axis in eosinophils promotes antitumor immunity through activation of type 1 T cell responsesIsabelle C Arnold, Mariela Artola-Boran, Alessandra Gurtner, et al.
Immunity|November 22, 2011
Cre-mediated cell ablation contests mast cell contribution in models of antibody- and T cell-mediated autoimmunityThorsten B Feyerabend, Anne Weiser, Annette Tietz, et al.
Plos One|August 5, 2009
Malarial hemozoin is a Nalp3 inflammasome activating danger signalCatherine Dostert, Greta Guarda, Jackeline F Romero, et al.
Cell Reports|April 27, 2025
Peroxisomes are critical for a unique metabolic demand and survival of alveolar macrophagesMai Matsushita, Jonathan Muri, Ivan Berest, et al.
Brain Research|October 18, 2002
Targeted inactivation of the neurotensin type 1 receptor reveals its role in body temperature control and feeding behavior but not in analgesiaAnne Remaury, Natalio Vita, Sylvain Gendreau, et al.
Nature Communications|September 7, 2019
Interleukin-36 cytokines alter the intestinal microbiome and can protect against obesity and metabolic dysfunctionEirini Giannoudaki, Yasmina E Hernandez-Santana, Kelly Mulfaul, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 3, 2006
Pituitary adenylate cyclase-activating polypeptide (PACAP) decreases ischemic neuronal cell death in association with IL-6Hirokazu Ohtaki, Tomoya Nakamachi, Kenji Dohi, et al.
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