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Samuel S Pappas

Showing results (1-10 of 28) with videos related to

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Movement Disorders : Official Journal of the Movement Disorder Society|January 6, 2022
Oligodendrocyte and Extracellular Matrix Contributions to Central Nervous System Motor Function: Implications for DystoniaDhananjay Yellajoshyula, Samuel S Pappas, William T Dauer
Handbook of Clinical Neurology|January 13, 2018
Inherited dystonias: clinical features and molecular pathwaysCorinne E Weisheit, Samuel S Pappas, William T Dauer
Dystonia (Lausanne, Switzerland)|March 6, 2023
Genetic evidence of aberrant striatal synaptic maturation and secretory pathway alteration in a dystonia mouse modelDhananjay Yellajoshyula, Sunday Opeyemi, William T Dauer, et al.
JCI Insight|February 22, 2021
CNS critical periods: implications for dystonia and other neurodevelopmental disordersJay Li, Sumin Kim, Samuel S Pappas, et al.
Neuropharmacology|May 25, 2011
Opioid-mediated regulation of A11 diencephalospinal dopamine neurons: pharmacological evidence of activation by morphineSamuel S Pappas, Tom Kennedy, John L Goudreau, et al.
Elife|March 24, 2020
TorsinB overexpression prevents abnormal twisting in DYT1 dystonia mouse modelsJay Li, Chun-Chi Liang, Samuel S Pappas, et al.
Frontiers in Neuroscience|December 5, 2025
Loss of Nocturnin increases neuronal viability in oxidative stress conditionsAnne E Ojo, Crystal Olivas-Rasmussen, Samuel S Pappas, et al.
Current Topics in Developmental Biology|June 21, 2014
Mouse models of neurodevelopmental disease of the basal ganglia and associated circuitsSamuel S Pappas, Daniel K Leventhal, Roger L Albin, et al.
Brain Research|May 9, 2008
Lack of D2 receptor mediated regulation of dopamine synthesis in A11 diencephalospinal neurons in male and female miceSamuel S Pappas, Bahareh Behrouz, Kelly L Janis, et al.
Human Molecular Genetics|November 30, 2017
TorsinA dysfunction causes persistent neuronal nuclear pore defectsSamuel S Pappas, Chun-Chi Liang, Sumin Kim, et al.
Pageof 3

Showing results (1-10 of 28) with videos related to

Sort By:
Pageof 3
Movement Disorders : Official Journal of the Movement Disorder Society|January 6, 2022
Oligodendrocyte and Extracellular Matrix Contributions to Central Nervous System Motor Function: Implications for DystoniaDhananjay Yellajoshyula, Samuel S Pappas, William T Dauer
Handbook of Clinical Neurology|January 13, 2018
Inherited dystonias: clinical features and molecular pathwaysCorinne E Weisheit, Samuel S Pappas, William T Dauer
Dystonia (Lausanne, Switzerland)|March 6, 2023
Genetic evidence of aberrant striatal synaptic maturation and secretory pathway alteration in a dystonia mouse modelDhananjay Yellajoshyula, Sunday Opeyemi, William T Dauer, et al.
JCI Insight|February 22, 2021
CNS critical periods: implications for dystonia and other neurodevelopmental disordersJay Li, Sumin Kim, Samuel S Pappas, et al.
Neuropharmacology|May 25, 2011
Opioid-mediated regulation of A11 diencephalospinal dopamine neurons: pharmacological evidence of activation by morphineSamuel S Pappas, Tom Kennedy, John L Goudreau, et al.
Elife|March 24, 2020
TorsinB overexpression prevents abnormal twisting in DYT1 dystonia mouse modelsJay Li, Chun-Chi Liang, Samuel S Pappas, et al.
Frontiers in Neuroscience|December 5, 2025
Loss of Nocturnin increases neuronal viability in oxidative stress conditionsAnne E Ojo, Crystal Olivas-Rasmussen, Samuel S Pappas, et al.
Current Topics in Developmental Biology|June 21, 2014
Mouse models of neurodevelopmental disease of the basal ganglia and associated circuitsSamuel S Pappas, Daniel K Leventhal, Roger L Albin, et al.
Brain Research|May 9, 2008
Lack of D2 receptor mediated regulation of dopamine synthesis in A11 diencephalospinal neurons in male and female miceSamuel S Pappas, Bahareh Behrouz, Kelly L Janis, et al.
Human Molecular Genetics|November 30, 2017
TorsinA dysfunction causes persistent neuronal nuclear pore defectsSamuel S Pappas, Chun-Chi Liang, Sumin Kim, et al.
Pageof 3