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Current Pathobiology Reports|September 4, 2013
Reversibility of Liver Fibrosis and Inactivation of Fibrogenic MyofibroblastsXiao Liu, Jun Xu, David A Brenner, et al.
Digestive Diseases (Basel, Switzerland)|June 23, 2016
New Developments on the Treatment of Liver FibrosisYukinori Koyama, Jun Xu, Xiao Liu, et al.
Hepatology (Baltimore, Md.)|April 5, 2013
Hepatic macrophages but not dendritic cells contribute to liver fibrosis by promoting the survival of activated hepatic stellate cells in miceJean-Philippe Pradere, Johannes Kluwe, Samuele De Minicis, et al.
Learning Health Systems|July 14, 2023
Developing a highly-reliable learning health systemRobert El-Kareh, David A Brenner, Christopher A Longhurst
Plos One|May 24, 2014
HCC development is associated to peripheral insulin resistance in a mouse model of NASHSamuele De Minicis, Laura Agostinelli, Chiara Rychlicki, et al.
Hepatology (Baltimore, Md.)|September 25, 2004
A dual reporter gene transgenic mouse demonstrates heterogeneity in hepatic fibrogenic cell populationsScott T Magness, Ramón Bataller, Liu Yang, et al.
Antioxidants & Redox Signaling|August 24, 2005
Liver fibrogenesis: a new role for the renin-angiotensin systemRamón Bataller, Pau Sancho-Bru, Pere Ginès, et al.
Liver International : Official Journal of the International Association for the Study of the Liver|July 13, 2011
Glucagon-like peptide-1 receptor activation stimulates hepatic lipid oxidation and restores hepatic signalling alteration induced by a high-fat diet in nonalcoholic steatohepatitisGianluca Svegliati-Baroni, Stefania Saccomanno, Chiara Rychlicki, et al.
Hepatology (Baltimore, Md.)|August 2, 2008
Hepatitis C virus-induced oxidative stress suppresses hepcidin expression through increased histone deacetylase activityKouichi Miura, Kojiro Taura, Yuzo Kodama, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|April 18, 2003
Salicylate enhances necrosis and apoptosis mediated by the mitochondrial permeability transitionKi-Wan Oh, Ting Qian, David A Brenner, et al.
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