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Research Square|March 17, 2025
Disturbed Flow Induces Reprogramming of Endothelial Cells to Immune-like and Foam Cells under Hypercholesterolemia during AtherogenesisChristian Park, Kyung In Baek, Ruei-Chun Hung, et al.
Cell Genomics|January 8, 2024
Multi-ancestry genetic analysis of gene regulation in coronary arteries prioritizes disease risk lociChani J Hodonsky, Adam W Turner, Mohammad Daud Khan, et al.
Circulation Research|August 30, 2023
Proteomic Atlas of Atherosclerosis: The Contribution of Proteoglycans to Sex Differences, Plaque Phenotypes, and OutcomesKonstantinos Theofilatos, Stefan Stojkovic, Maria Hasman, et al.
Atherosclerosis|December 6, 2021
Enhanced single-cell RNA-seq workflow reveals coronary artery disease cellular cross-talk and candidate drug targetsWei Feng Ma, Chani J Hodonsky, Adam W Turner, et al.
Medrxiv : the Preprint Server for Health Sciences|February 24, 2023
Integrative multi-ancestry genetic analysis of gene regulation in coronary arteries prioritizes disease risk lociChani J Hodonsky, Adam W Turner, Mohammad Daud Khan, et al.
Science Advances|May 1, 2023
Druggable proteins influencing cardiac structure and function: Implications for heart failure therapies and cancer cardiotoxicityAmand F Schmidt, Mimount Bourfiss, Abdulrahman Alasiri, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|April 16, 2016
Human Validation of Genes Associated With a Murine Atherosclerotic PhenotypeGerard Pasterkamp, Sander W van der Laan, Saskia Haitjema, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|April 18, 2024
Atherosclerotic Plaque Epigenetic Age Acceleration Predicts a Poor Prognosis and Is Associated With Endothelial-to-Mesenchymal Transition in HumansErnest Diez Benavente, Robin J G Hartman, Tim R Sakkers, et al.
Circulation Research|February 5, 2026
Atherosclerotic Fibrous Plaques in Women Present ECM Remodeling Linked to TGF-βTim R Sakkers, Eloi Mili, Hanna Winter, et al.
Scientific Reports|February 23, 2018
Network analysis of coronary artery disease risk genes elucidates disease mechanisms and druggable targetsHarri Lempiäinen, Ingrid Brænne, Tom Michoel, et al.
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