Showing results (1-10 of 11) with videos related to

Sort By:
Pageof 2
International Journal of Molecular Sciences|October 13, 2021
Phospho-Tau and Chromatin Landscapes in Early and Late Alzheimer's DiseaseLaura Gil, Sandra A Niño, Carmen Guerrero, et al.
Neuroscience Letters|February 21, 2021
Aging and Alzheimer's disease connection: Nuclear Tau and lamin ALaura Gil, Sandra A Niño, Gabriela Capdeville, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|March 5, 2026
Mitochondrial complex-derived ROS induces lysosomal dysfunction and impairs autophagic flux in human cells carrying the APOE4 alleleSandra A Niño, Oskar Barrios-Camus, Eduardo Silva-Pavez, et al.
Toxicology and Applied Pharmacology|March 16, 2020
Demyelination associated with chronic arsenic exposure in Wistar ratsSandra A Niño, Erika Chi-Ahumada, Juan Ortíz, et al.
International Journal of Molecular Sciences|March 12, 2020
Perinuclear Lamin A and Nucleoplasmic Lamin B2 Characterize Two Types of Hippocampal Neurons through Alzheimer's Disease ProgressionLaura Gil, Sandra A Niño, Erika Chi-Ahumada, et al.
Brain Research|December 1, 2021
Life-long arsenic exposure damages the microstructure of the rat hippocampusSandra A Niño, Erika Chi-Ahumada, Leticia Carrizales, et al.
International Journal of Molecular Sciences|November 11, 2022
Pathological Nuclear Hallmarks in Dentate Granule Cells of Alzheimer's Patients: A Biphasic Regulation of NeurogenesisLaura Gil, Erika Chi-Ahumada, Sandra A Niño, et al.
Neurotoxicity Research|September 17, 2021
Cortical Synaptic Reorganization Under Chronic Arsenic ExposureSandra A Niño, Nallely Vázquez-Hernández, Jaime Arevalo-Villalobos, et al.
Neurochemical Research|January 21, 2026
Nuclear and Cytoplasmic Mouse ID Associated 1 (MIDA1) Protein Varies in Neurons and Astrocytes Across the Different Hippocampal Regions and Exhibits Age-Related ChangesAres Orlando Cuellar-Santoyo, Victor Manuel Ruiz-Rodríguez, Araceli Patrón-Soberano, et al.
Npj Aging|February 21, 2025
Calcium (Ca2+) fluxes at mitochondria-ER contact sites (MERCS) are a new target of senolysis in therapy-induced senescence (TIS)Andrea Puebla-Huerta, Hernán Huerta, Camila Quezada-Gutierez, et al.
Pageof 2