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The Journal of Physiology|July 16, 2005
Beta-scorpion toxin effects suggest electrostatic interactions in domain II of voltage-dependent sodium channelsMassimo Mantegazza, Sandrine Cestèle
Neuroscience Letters|November 14, 2017
Pathophysiological mechanisms of migraine and epilepsy: Similarities and differencesMassimo Mantegazza, Sandrine Cestèle
Physiological Reviews|March 26, 2021
Sodium channelopathies of skeletal muscle and brainMassimo Mantegazza, Sandrine Cestèle, William A Catterall
Proceedings of the National Academy of Sciences of the United States of America|October 9, 2013
Nonfunctional NaV1.1 familial hemiplegic migraine mutant transformed into gain of function by partial rescue of folding defectsSandrine Cestèle, Emanuele Schiavon, Raffaella Rusconi, et al.
Neuropharmacology|June 3, 2017
Post-translational dysfunctions in channelopathies of the nervous systemBenedetta Terragni, Paolo Scalmani, Silvana Franceschetti, et al.
Journal of Neurochemistry|September 1, 2023
Voltage-gated sodium channels in genetic epilepsy: up and down of excitabilityEvgeniia Rusina, Martina Simonti, Fabrice Duprat, et al.
Frontiers in Molecular Neuroscience|July 25, 2018
Gain of Function for the SCN1A/hNav1.1-L1670W Mutation Responsible for Familial Hemiplegic MigraineSandra Dhifallah, Eric Lancaster, Shana Merrill, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 18, 2008
Self-limited hyperexcitability: functional effect of a familial hemiplegic migraine mutation of the Nav1.1 (SCN1A) Na+ channelSandrine Cestèle, Paolo Scalmani, Raffaella Rusconi, et al.
The Journal of Biological Chemistry|May 9, 2006
Structure and function of the voltage sensor of sodium channels probed by a beta-scorpion toxinSandrine Cestèle, Vladimir Yarov-Yarovoy, Yusheng Qu, et al.
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