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Cell Reports|September 8, 2023
A co-opted endogenous retroviral envelope promotes cell survival by controlling CTR1-mediated copper transport and homeostasisSandrine Tury, Lise Chauveau, Arnaud Lecante, et al.
Journal of Virology|June 2, 2022
Identification of Copper Transporter 1 as a Receptor for Feline Endogenous Retrovirus ERV-DC14Sandrine Tury, Donatella Giovannini, Svilena Ivanova, et al.
Nature Communications|January 15, 2025
Structural basis of phosphate export by human XPR1Qixian He, Ran Zhang, Sandrine Tury, et al.
Oncotarget|November 12, 2016
Combination of COX-2 expression and PIK3CA mutation as prognostic and predictive markers for celecoxib treatment in breast cancerSandrine Tury, Véronique Becette, Franck Assayag, et al.
The Journal of Biological Chemistry|May 13, 2020
Interplay between primary familial brain calcification-associated SLC20A2 and XPR1 phosphate transporters requires inositol polyphosphates for control of cellular phosphate homeostasisUriel López-Sánchez, Sandrine Tury, Gaël Nicolas, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|February 1, 2019
Celecoxib With Neoadjuvant Chemotherapy for Breast Cancer Might Worsen Outcomes Differentially by COX-2 Expression and ER Status: Exploratory Analysis of the REMAGUS02 TrialAnne-Sophie Hamy, Sandrine Tury, Xiaofei Wang, et al.
The Journal of Pathology|June 8, 2018
The iron chelator deferasirox synergises with chemotherapy to treat triple-negative breast cancersSandrine Tury, Franck Assayag, Florian Bonin, et al.
BMC Biology|October 5, 2018
VOPP1 promotes breast tumorigenesis by interacting with the tumor suppressor WWOXFlorian Bonin, Karim Taouis, Paula Azorin, et al.
Movement Disorders : Official Journal of the Movement Disorder Society|June 8, 2020
Haploinsufficiency of the Primary Familial Brain Calcification Gene SLC20A2 Mediated by Disruption of a Regulatory ElementKévin Cassinari, Anne Rovelet-Lecrux, Sandrine Tury, et al.
Nature Communications|March 14, 2024
Biallelic NAA60 variants with impaired n-terminal acetylation capacity cause autosomal recessive primary familial brain calcificationsViorica Chelban, Henriette Aksnes, Reza Maroofian, et al.
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