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Sandy Chang

Showing results (41-50 of 79) with videos related to

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Nature Communications|September 18, 2021
Distinct functions of POT1 proteins contribute to the regulation of telomerase recruitment to telomeresPeili Gu, Shuting Jia, Taylor Takasugi, et al.
The EMBO Journal|July 1, 2010
The function of classical and alternative non-homologous end-joining pathways in the fusion of dysfunctional telomeresRekha Rai, Hong Zheng, Hua He, et al.
The EMBO Journal|April 26, 2012
CTC1 deletion results in defective telomere replication, leading to catastrophic telomere loss and stem cell exhaustionPeili Gu, Jin-Na Min, Yang Wang, et al.
The Journal of Nursing Administration|May 2, 2002
Advanced practice nursing. Acute care model in progressRita Whitcomb, Sandra Wilson, Sandy Chang-Dawkins, et al.
Diagnosis (Berlin, Germany)|March 16, 2019
Internal medicine residents' evaluation of fevers overnightJessica Howard-Anderson, Kristin E Schwab, Sandy Chang, et al.
Cancer Cell|September 3, 2002
Telomere dysfunction provokes regional amplification and deletion in cancer genomesRónán C O'Hagan, Sandy Chang, Richard S Maser, et al.
The EMBO Journal|October 21, 2006
POT1b protects telomeres from end-to-end chromosomal fusions and aberrant homologous recombinationHua He, Asha S Multani, Wilfredo Cosme-Blanco, et al.
EMBO Reports|March 31, 2007
Telomere dysfunction suppresses spontaneous tumorigenesis in vivo by initiating p53-dependent cellular senescenceWilfredo Cosme-Blanco, Mei-Feng Shen, Alexander J F Lazar, et al.
Cancer Research|August 3, 2007
Overexpression of the low molecular weight cyclin E in transgenic mice induces metastatic mammary carcinomas through the disruption of the ARF-p53 pathwaySaid Akli, Carolyn S Van Pelt, Tuyen Bui, et al.
The EMBO Journal|October 20, 2007
Dysfunctional telomeres activate an ATM-ATR-dependent DNA damage response to suppress tumorigenesisXiaolan Guo, Yibin Deng, Yahong Lin, et al.
Pageof 8

Showing results (41-50 of 79) with videos related to

Sort By:
Pageof 8
Nature Communications|September 18, 2021
Distinct functions of POT1 proteins contribute to the regulation of telomerase recruitment to telomeresPeili Gu, Shuting Jia, Taylor Takasugi, et al.
The EMBO Journal|July 1, 2010
The function of classical and alternative non-homologous end-joining pathways in the fusion of dysfunctional telomeresRekha Rai, Hong Zheng, Hua He, et al.
The EMBO Journal|April 26, 2012
CTC1 deletion results in defective telomere replication, leading to catastrophic telomere loss and stem cell exhaustionPeili Gu, Jin-Na Min, Yang Wang, et al.
The Journal of Nursing Administration|May 2, 2002
Advanced practice nursing. Acute care model in progressRita Whitcomb, Sandra Wilson, Sandy Chang-Dawkins, et al.
Diagnosis (Berlin, Germany)|March 16, 2019
Internal medicine residents' evaluation of fevers overnightJessica Howard-Anderson, Kristin E Schwab, Sandy Chang, et al.
Cancer Cell|September 3, 2002
Telomere dysfunction provokes regional amplification and deletion in cancer genomesRónán C O'Hagan, Sandy Chang, Richard S Maser, et al.
The EMBO Journal|October 21, 2006
POT1b protects telomeres from end-to-end chromosomal fusions and aberrant homologous recombinationHua He, Asha S Multani, Wilfredo Cosme-Blanco, et al.
EMBO Reports|March 31, 2007
Telomere dysfunction suppresses spontaneous tumorigenesis in vivo by initiating p53-dependent cellular senescenceWilfredo Cosme-Blanco, Mei-Feng Shen, Alexander J F Lazar, et al.
Cancer Research|August 3, 2007
Overexpression of the low molecular weight cyclin E in transgenic mice induces metastatic mammary carcinomas through the disruption of the ARF-p53 pathwaySaid Akli, Carolyn S Van Pelt, Tuyen Bui, et al.
The EMBO Journal|October 20, 2007
Dysfunctional telomeres activate an ATM-ATR-dependent DNA damage response to suppress tumorigenesisXiaolan Guo, Yibin Deng, Yahong Lin, et al.
Pageof 8