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Cell Reports|July 26, 2023
mTOR inhibition reprograms cellular proteostasis by regulating eIF3D-mediated selective mRNA translation and promotes cell phenotype switchingSejeong Shin, Min-Joon Han, Mark P Jedrychowski, et al.
Current Biology : CB|September 16, 2014
The mTORC1/S6K1 pathway regulates glutamine metabolism through the eIF4B-dependent control of c-Myc translationAlfredo Csibi, Gina Lee, Sang-Oh Yoon, et al.
Molecular Cell|June 2, 2010
Glucose addiction of TSC null cells is caused by failed mTORC1-dependent balancing of metabolic demand with supplyAndrew Y Choo, Sang Gyun Kim, Matthew G Vander Heiden, et al.
Molecular Cell|September 19, 2025
mTOR inhibition reprograms cellular lipid homeostasis by inducing alternative lipid uptake and promoting cholesterol transportSejeong Shin, Min-Joon Han, Ishika Patel, et al.
Cancer Research|February 15, 2018
RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in MelanomaThibault Houles, Simon-Pierre Gravel, Geneviève Lavoie, et al.
Experimental Cell Research|March 30, 2019
Metabolic switching in pluripotent stem cells reorganizes energy metabolism and subcellular organellesCarla O'Reilly, Ji-Hoon Cho, Qian Qi, et al.
Science (New York, N.Y.)|June 11, 2011
Phosphoproteomic analysis identifies Grb10 as an mTORC1 substrate that negatively regulates insulin signalingYonghao Yu, Sang-Oh Yoon, George Poulogiannis, et al.
Molecular Cell|August 1, 2017
Focal Adhesion- and IGF1R-Dependent Survival and Migratory Pathways Mediate Tumor Resistance to mTORC1/2 InhibitionSang-Oh Yoon, Sejeong Shin, Florian A Karreth, et al.
Liver Transplantation : Official Publication of the American Association for the Study of Liver Diseases and the International Liver Transplantation Society|April 7, 2023
Improved recurrence-free survival in patients with HCC with post-transplant plasma exchangeNamkee Oh, Jinsoo Rhu, Jong Man Kim, et al.
Molecular Cell|June 5, 2018
mTORC1 Promotes Metabolic Reprogramming by the Suppression of GSK3-Dependent Foxk1 PhosphorylationLong He, Ana P Gomes, Xin Wang, et al.
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