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Sanja Sever

Showing results (31-40 of 41) with videos related to

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Kidney International|February 7, 2023
The small GTPase regulatory protein Rac1 drives podocyte injury independent of cationic channel protein TRPC5Onur K Polat, Elena Isaeva, Yashwanth R Sudhini, et al.
Nature Medicine|December 13, 2016
Bone marrow-derived immature myeloid cells are a main source of circulating suPAR contributing to proteinuric kidney diseaseEunsil Hahm, Changli Wei, Isabel Fernandez, et al.
The Journal of Biological Chemistry|March 8, 2005
The low density lipoprotein receptor-related protein (LRP) is a novel beta-secretase (BACE1) substrateChristine A F von Arnim, Ayae Kinoshita, Ithan D Peltan, et al.
Nature Medicine|May 12, 2015
Pharmacological targeting of actin-dependent dynamin oligomerization ameliorates chronic kidney disease in diverse animal modelsMario Schiffer, Beina Teng, Changkyu Gu, et al.
The Journal of Clinical Investigation|February 8, 2019
uPAR isoform 2 forms a dimer and induces severe kidney disease in miceChangli Wei, Jing Li, Brian D Adair, et al.
JAMA Pediatrics|September 6, 2017
Association of Serum Soluble Urokinase Receptor Levels With Progression of Kidney Disease in ChildrenFranz Schaefer, Howard Trachtman, Elke Wühl, et al.
The New England Journal of Medicine|January 30, 2020
Soluble Urokinase Receptor and Acute Kidney InjurySalim S Hayek, David E Leaf, Ayman Samman Tahhan, et al.
Medrxiv : the Preprint Server for Health Sciences|April 2, 2024
Recessive variants in the intergenic <i>NOS1AP-C1orf226</i> locus cause monogenic kidney disease responsive to anti-proteinuric treatmentFlorian Buerger, Daanya Salmanullah, Lorrin Liang, et al.
The Journal of Clinical Investigation|September 14, 2011
CD2AP in mouse and human podocytes controls a proteolytic program that regulates cytoskeletal structure and cellular survivalSuma Yaddanapudi, Mehmet M Altintas, Andreas D Kistler, et al.
Nature Medicine|June 27, 2017
A tripartite complex of suPAR, APOL1 risk variants and α<sub>v</sub>β<sub>3</sub> integrin on podocytes mediates chronic kidney diseaseSalim S Hayek, Kwi Hye Koh, Morgan E Grams, et al.
Pageof 5

Showing results (31-40 of 41) with videos related to

Sort By:
Pageof 5
Kidney International|February 7, 2023
The small GTPase regulatory protein Rac1 drives podocyte injury independent of cationic channel protein TRPC5Onur K Polat, Elena Isaeva, Yashwanth R Sudhini, et al.
Nature Medicine|December 13, 2016
Bone marrow-derived immature myeloid cells are a main source of circulating suPAR contributing to proteinuric kidney diseaseEunsil Hahm, Changli Wei, Isabel Fernandez, et al.
The Journal of Biological Chemistry|March 8, 2005
The low density lipoprotein receptor-related protein (LRP) is a novel beta-secretase (BACE1) substrateChristine A F von Arnim, Ayae Kinoshita, Ithan D Peltan, et al.
Nature Medicine|May 12, 2015
Pharmacological targeting of actin-dependent dynamin oligomerization ameliorates chronic kidney disease in diverse animal modelsMario Schiffer, Beina Teng, Changkyu Gu, et al.
The Journal of Clinical Investigation|February 8, 2019
uPAR isoform 2 forms a dimer and induces severe kidney disease in miceChangli Wei, Jing Li, Brian D Adair, et al.
JAMA Pediatrics|September 6, 2017
Association of Serum Soluble Urokinase Receptor Levels With Progression of Kidney Disease in ChildrenFranz Schaefer, Howard Trachtman, Elke Wühl, et al.
The New England Journal of Medicine|January 30, 2020
Soluble Urokinase Receptor and Acute Kidney InjurySalim S Hayek, David E Leaf, Ayman Samman Tahhan, et al.
Medrxiv : the Preprint Server for Health Sciences|April 2, 2024
Recessive variants in the intergenic <i>NOS1AP-C1orf226</i> locus cause monogenic kidney disease responsive to anti-proteinuric treatmentFlorian Buerger, Daanya Salmanullah, Lorrin Liang, et al.
The Journal of Clinical Investigation|September 14, 2011
CD2AP in mouse and human podocytes controls a proteolytic program that regulates cytoskeletal structure and cellular survivalSuma Yaddanapudi, Mehmet M Altintas, Andreas D Kistler, et al.
Nature Medicine|June 27, 2017
A tripartite complex of suPAR, APOL1 risk variants and α<sub>v</sub>β<sub>3</sub> integrin on podocytes mediates chronic kidney diseaseSalim S Hayek, Kwi Hye Koh, Morgan E Grams, et al.
Pageof 5