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Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|December 31, 2016
Phase III, Double-Blind, Randomized Trial That Compared Maintenance Lapatinib Versus Placebo After First-Line Chemotherapy in Patients With Human Epidermal Growth Factor Receptor 1/2-Positive Metastatic Bladder CancerThomas Powles, Robert A Huddart, Tony Elliott, et al.Nature|June 23, 2012
Proto-genes and de novo gene birthAnne-Ruxandra Carvunis, Thomas Rolland, Ilan Wapinski, et al.BMJ Global Health|May 15, 2024
Long-term healthcare utilisation, costs and quality of life after invasive group B Streptococcus disease: a cohort study in five low-income and middle-income countriesFarah Seedat, Simon Procter, Ziyaad Dangor, et al.The Lancet. Infectious Diseases|December 10, 2014
Burden of hepatitis C virus disease and access to hepatitis C virus services in people who inject drugs in India: a cross-sectional studySunil Suhas Solomon, Shruti H Mehta, Aylur K Srikrishnan, et al.Nano Letters|August 10, 2006
Volatile organic compound detection using nanostructured copolymersBo Li, Genevieve Sauvé, Mihaela C Iovu, et al.International Immunology|June 6, 2018
Anti-leukemic effects of all-trans retinoic acid in combination with Daratumumab in acute myeloid leukemiaNathaniel J Buteyn, Kavin Fatehchand, Ramasamy Santhanam, et al.Cell Death & Disease|April 22, 2020
Transient receptor potential ion channel TRPM2 promotes AML proliferation and survival through modulation of mitochondrial function, ROS, and autophagyShu-Jen Chen, Lei Bao, Kerry Keefer, et al.Nature Communications|August 31, 2018
FOXD1-dependent MICU1 expression regulates mitochondrial activity and cell differentiationSanthanam Shanmughapriya, Dhanendra Tomar, Zhiwei Dong, et al.Science Signaling|March 5, 2015
Ca2+ signals regulate mitochondrial metabolism by stimulating CREB-mediated expression of the mitochondrial Ca2+ uniporter gene MCUSanthanam Shanmughapriya, Sudarsan Rajan, Nicholas E Hoffman, et al.Cancer Cell|November 16, 2005
The tumor suppressor PP2A is functionally inactivated in blast crisis CML through the inhibitory activity of the BCR/ABL-regulated SET proteinPaolo Neviani, Ramasamy Santhanam, Rossana Trotta, et al.Pageof 132