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Journal of Anatomy|October 30, 2015
Quantitative imaging of tissue sections using infrared scanning technologySamantha L Eaton, Elizabeth Cumyn, Declan King, et al.Open Biology|June 12, 2015
Warburg Micro syndrome is caused by RAB18 deficiency or dysregulationMark T Handley, Sarah M Carpanini, Girish R Mali, et al.Frontiers in Neuroscience|May 22, 2025
TREM2 supports neuronal protection and microglial reactivity without an effect on misfolded protein deposition in chronic neurodegenerative prion diseaseSarah M Carpanini, Barry M Bradford, Alessio Alfieri, et al.Journal of Neuroinflammation|July 21, 2023
Alzheimer's disease-associated complement gene variants influence plasma complement protein levelsAurora Veteleanu, Joshua Stevenson-Hoare, Samuel Keat, et al.Acta Neuropathologica Communications|July 6, 2022
Terminal complement pathway activation drives synaptic loss in Alzheimer's disease modelsSarah M Carpanini, Megan Torvell, Ryan J Bevan, et al.Brain, Behavior, and Immunity|March 20, 2025
The schizophrenia-associated gene CSMD1 encodes a complement classical pathway inhibitor predominantly expressed by astrocytes and at synapses in mice and humansRobert A J Byrne, Jacqui Nimmo, Megan Torvell, et al.Journal of Cell Science|January 5, 2017
Sideroflexin 3 is an α-synuclein-dependent mitochondrial protein that regulates synaptic morphologyInês S Amorim, Laura C Graham, Roderick N Carter, et al.Disease Models & Mechanisms|April 26, 2014
A novel mouse model of Warburg Micro syndrome reveals roles for RAB18 in eye development and organisation of the neuronal cytoskeletonSarah M Carpanini, Lisa McKie, Derek Thomson, et al.Elife|July 22, 2017
Pro-death NMDA receptor signaling is promoted by the GluN2B C-terminus independently of Dapk1Jamie McQueen, Tomás J Ryan, Sean McKay, et al.Stem Cell Reports|July 4, 2025
Modeling common Alzheimer's disease with high and low polygenic risk in human iPSC: A large-scale research resourceEmily Maguire, Jincy Winston, Sarah H Ellwood, et al.Pageof 3