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Sarah Van Veen

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Proceedings of the National Academy of Sciences of the United States of America|November 24, 2020

ATP13A2-mediated endo-lysosomal polyamine export counters mitochondrial oxidative stressStephanie Vrijsen, Laura Besora-Casals, Sarah van Veen, et al.

Nature|January 31, 2020

ATP13A2 deficiency disrupts lysosomal polyamine exportSarah van Veen, Shaun Martin, Chris Van den Haute, et al.

Acta Neuropathologica|March 16, 2020

Mutated ATP10B increases Parkinson's disease risk by compromising lysosomal glucosylceramide exportShaun Martin, Stefanie Smolders, Chris Van den Haute, et al.

Brain : a Journal of Neurology|February 1, 2017

Loss-of-function mutations in the ATP13A2/PARK9 gene cause complicated hereditary spastic paraplegia (SPG78)Alejandro Estrada-Cuzcano, Shaun Martin, Teodora Chamova, et al.

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Search research articles

Showing results (11-20 of 14) with videos related to

Sort By:

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You have reached the last page of results.This site can display upto 14 results.

Proceedings of the National Academy of Sciences of the United States of America|November 24, 2020

ATP13A2-mediated endo-lysosomal polyamine export counters mitochondrial oxidative stressStephanie Vrijsen, Laura Besora-Casals, Sarah van Veen, et al.

Nature|January 31, 2020

ATP13A2 deficiency disrupts lysosomal polyamine exportSarah van Veen, Shaun Martin, Chris Van den Haute, et al.

Acta Neuropathologica|March 16, 2020

Mutated ATP10B increases Parkinson's disease risk by compromising lysosomal glucosylceramide exportShaun Martin, Stefanie Smolders, Chris Van den Haute, et al.

Brain : a Journal of Neurology|February 1, 2017

Loss-of-function mutations in the ATP13A2/PARK9 gene cause complicated hereditary spastic paraplegia (SPG78)Alejandro Estrada-Cuzcano, Shaun Martin, Teodora Chamova, et al.

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