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Biochemical and Biophysical Research Communications|November 22, 2024
GLP-1 receptor signaling restores aquaporin 4 subcellular polarization in reactive astrocytes and promotes amyloid β clearance in a mouse model of Alzheimer's diseaseKana Sasaki, Hiroki Fujita, Takehiro Sato, et al.The Journal of Clinical Investigation|November 15, 2023
Intracytoplasmic sperm injection induces transgenerational abnormalities in miceMito Kanatsu-Shinohara, Yusuke Shiromoto, Narumi Ogonuki, et al.Inflammation and Regeneration|July 9, 2025
Mutation of the histone demethylase Gasc1 causes ASD-like symptoms in miceTetsushi Kagawa, Yuhei Yamaguchi, Yasuhiro Kokubu, et al.Scientific Reports|August 27, 2020
Impairment of spatial memory accuracy improved by Cbr1 copy number resumption and GABAB receptor-dependent enhancement of synaptic inhibition in Down syndrome model miceFumiko Arima-Yoshida, Matthieu Raveau, Atsushi Shimohata, et al.Iscience|August 22, 2022
Humanized substitutions of Vmat1 in mice alter amygdala-dependent behaviors associated with the evolution of anxietyDaiki X Sato, Yukiko U Inoue, Nahoko Kuga, et al.Brain Communications|September 15, 2022
Deficiency of CHAMP1, a gene related to intellectual disability, causes impaired neuronal development and a mild behavioural phenotypeMasayoshi Nagai, Kenji Iemura, Takako Kikkawa, et al.Molecular Neurobiology|May 14, 2025
Chronic Hyponatremia Potentiates Innate Anxiety-Like Behaviors Through the Dysfunction of Monoaminergic Neurons in MiceHaruki Fujisawa, Nobuhiko Magara, Shogo Nakayama, et al.Experimental Neurology|March 25, 2017
Ts1Cje Down syndrome model mice exhibit environmental stimuli-triggered locomotor hyperactivity and sociability concurrent with increased flux through central dopamine and serotonin metabolismAtsushi Shimohata, Keiichi Ishihara, Satoko Hattori, et al.Nature Communications|December 15, 2015
A CDC42EP4/septin-based perisynaptic glial scaffold facilitates glutamate clearanceNatsumi Ageta-Ishihara, Maya Yamazaki, Kohtarou Konno, et al.Scientific Reports|February 8, 2013
Post-natal treatment by a blood-brain-barrier permeable calpain inhibitor, SNJ1945 rescued defective function in lissencephalyShiori Toba, Yasuhisa Tamura, Kanako Kumamoto, et al.Pageof 7