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The Biochemical Journal
|
July 10, 2024
E3 ligases: a ubiquitous link between DNA repair, DNA replication and human disease
Anoop S Chauhan, Satpal S Jhujh, Grant S Stewart
Ageing Research Reviews
|
February 22, 2023
Discovery of a new hereditary RECQ helicase disorder RECON syndrome positions the replication stress response and genome homeostasis as centrally important processes in aging and age-related disease
Arindam Datta, Joshua A Sommers, Satpal S Jhujh, et al.
Molecular Cell
|
October 11, 2023
The structural mechanism of dimeric DONSON in replicative helicase activation
Milos A Cvetkovic, Paolo Passaretti, Agata Butryn, et al.
Cancer Research
|
January 14, 2022
Cancer-Associated SF3B1 Mutations Confer a BRCA-Like Cellular Phenotype and Synthetic Lethality to PARP Inhibitors
Katrina M Lappin, Eliana M Barros, Satpal S Jhujh, et al.
Nature Communications
|
April 14, 2025
PIN1-SUMO2/3 motif suppresses excessive RNF168 chromatin accumulation and ubiquitin signaling to promote IR resistance
Anoop S Chauhan, Matthew J W Mackintosh, Joseph Cassar, et al.
Biorxiv : the Preprint Server for Biology
|
September 16, 2024
USP37 prevents premature disassembly of stressed replisomes by TRAIP
Olga V Kochenova, Giuseppina D'Alessandro, Domenic Pilger, et al.
Nature Communications
|
June 18, 2025
USP37 prevents premature disassembly of stressed replisomes by TRAIP
Olga V Kochenova, Giuseppina D'Alessandro, Domenic Pilger, et al.
Nucleic Acids Research
|
October 24, 2024
PCNA-binding activity separates RNF168 functions in DNA replication and DNA double-stranded break signaling
Yang Yang, Deepika Jayaprakash, Satpal S Jhujh, et al.
EMBO Molecular Medicine
|
December 19, 2025
Loss of CTLH component MAEA impairs DNA repair and replication and leads to developmental delay
Søren H Hough, Satpal S Jhujh, Samah W Awwad, et al.
The Journal of Clinical Investigation
|
January 13, 2022
RECON syndrome is a genome instability disorder caused by mutations in the DNA helicase RECQL1
Bassam Abu-Libdeh, Satpal S Jhujh, Srijita Dhar, et al.
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Search research articles
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Showing results (1-10 of 12) with videos related to
Sort By:
Page
of 2
The Biochemical Journal
|
July 10, 2024
E3 ligases: a ubiquitous link between DNA repair, DNA replication and human disease
Anoop S Chauhan, Satpal S Jhujh, Grant S Stewart
Ageing Research Reviews
|
February 22, 2023
Discovery of a new hereditary RECQ helicase disorder RECON syndrome positions the replication stress response and genome homeostasis as centrally important processes in aging and age-related disease
Arindam Datta, Joshua A Sommers, Satpal S Jhujh, et al.
Molecular Cell
|
October 11, 2023
The structural mechanism of dimeric DONSON in replicative helicase activation
Milos A Cvetkovic, Paolo Passaretti, Agata Butryn, et al.
Cancer Research
|
January 14, 2022
Cancer-Associated SF3B1 Mutations Confer a BRCA-Like Cellular Phenotype and Synthetic Lethality to PARP Inhibitors
Katrina M Lappin, Eliana M Barros, Satpal S Jhujh, et al.
Nature Communications
|
April 14, 2025
PIN1-SUMO2/3 motif suppresses excessive RNF168 chromatin accumulation and ubiquitin signaling to promote IR resistance
Anoop S Chauhan, Matthew J W Mackintosh, Joseph Cassar, et al.
Biorxiv : the Preprint Server for Biology
|
September 16, 2024
USP37 prevents premature disassembly of stressed replisomes by TRAIP
Olga V Kochenova, Giuseppina D'Alessandro, Domenic Pilger, et al.
Nature Communications
|
June 18, 2025
USP37 prevents premature disassembly of stressed replisomes by TRAIP
Olga V Kochenova, Giuseppina D'Alessandro, Domenic Pilger, et al.
Nucleic Acids Research
|
October 24, 2024
PCNA-binding activity separates RNF168 functions in DNA replication and DNA double-stranded break signaling
Yang Yang, Deepika Jayaprakash, Satpal S Jhujh, et al.
EMBO Molecular Medicine
|
December 19, 2025
Loss of CTLH component MAEA impairs DNA repair and replication and leads to developmental delay
Søren H Hough, Satpal S Jhujh, Samah W Awwad, et al.
The Journal of Clinical Investigation
|
January 13, 2022
RECON syndrome is a genome instability disorder caused by mutations in the DNA helicase RECQL1
Bassam Abu-Libdeh, Satpal S Jhujh, Srijita Dhar, et al.
Page
of 2