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Chemico-Biological Interactions|March 13, 2019
Attenuation of the effects of oxidative stress by the MAO-inhibiting antidepressant and carbonyl scavenger phenelzineGlen Baker, Dmitriy Matveychuk, Erin M MacKenzie, et al.
The Journal of Biological Chemistry|December 20, 2012
Role of cathepsin D in U18666A-induced neuronal cell death: potential implication in Niemann-Pick type C disease pathogenesisAsha Amritraj, Yanlin Wang, Timothy J Revett, et al.
The International Journal of Social Psychiatry|November 3, 2022
Mental health disorders in English newspapers of India: A retrospective studyGervan J Arneaud, Anindya Kar, Sunrit Majumder, et al.
Endocrinology|August 21, 2012
Single-transmembrane domain IGF-II/M6P receptor: potential interaction with G protein and its association with cholesterol-rich membrane domainsAsha Amritraj, Elena I Posse de Chaves, Cheryl Hawkes, et al.
Human Molecular Genetics|August 8, 2012
Mutant human APP exacerbates pathology in a mouse model of NPC and its reversal by a β-cyclodextrinMahua Maulik, Bibaswan Ghoshal, John Kim, et al.
Molecular Neurobiology|May 27, 2019
Proteasomal Inhibition Redirects the PrP-Like Shadoo Protein to the NucleusSang-Gyun Kang, Charles E Mays, Nathalie Daude, et al.
International Journal of Biological Macromolecules|August 28, 2022
Native PLGA nanoparticles regulate APP metabolism and protect neurons against β-amyloid toxicity: Potential significance in Alzheimer's disease pathologyQi Wu, Govindarajan Karthivashan, Maryam Nakhaei-Nejad, et al.
ACS Chemical Neuroscience|November 17, 2025
Native PEG-PLGA Attenuates β-Amyloid Aggregation and Toxicity under In Vitro ConditionsMallesh Rathnam, Haley Hunter, Pallabi Sil Paul, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 13, 2006
Single transmembrane domain insulin-like growth factor-II/mannose-6-phosphate receptor regulates central cholinergic function by activating a G-protein-sensitive, protein kinase C-dependent pathwayCheryl Hawkes, Jack H Jhamandas, Kim H Harris, et al.
The American Journal of Pathology|November 7, 2009
Increased activity and altered subcellular distribution of lysosomal enzymes determine neuronal vulnerability in Niemann-Pick type C1-deficient miceAsha Amritraj, Kyle Peake, Anitha Kodam, et al.
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