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Free Radical Biology & Medicine
|
November 25, 2017
KRIT1 loss-of-function induces a chronic Nrf2-mediated adaptive homeostasis that sensitizes cells to oxidative stress: Implication for Cerebral Cavernous Malformation disease
Cinzia Antognelli, Eliana Trapani, Simona Delle Monache, et al.
Scientific Reports
|
August 17, 2017
Up-regulation of NADPH oxidase-mediated redox signaling contributes to the loss of barrier function in KRIT1 deficient endothelium
Luca Goitre, Peter V DiStefano, Andrea Moglia, et al.
Antioxidants (Basel, Switzerland)
|
January 20, 2019
KRIT1 Loss-Of-Function Associated with Cerebral Cavernous Malformation Disease Leads to Enhanced <i>S</i>-Glutathionylation of Distinct Structural and Regulatory Proteins
Laura Cianfruglia, Andrea Perrelli, Claudia Fornelli, et al.
Brain Pathology (Zurich, Switzerland)
|
October 30, 2010
Mutation analysis of CCM1, CCM2 and CCM3 genes in a cohort of Italian patients with cerebral cavernous malformation
Rosalia D'Angelo, Valeria Marini, Carmela Rinaldi, et al.
International Journal of Molecular Sciences
|
October 9, 2019
KRIT1 Deficiency Promotes Aortic Endothelial Dysfunction
Francesco Vieceli Dalla Sega, Raffaella Mastrocola, Giorgio Aquila, et al.
Nature
|
June 11, 2013
EndMT contributes to the onset and progression of cerebral cavernous malformations
Luigi Maddaluno, Noemi Rudini, Roberto Cuttano, et al.
EMBO Molecular Medicine
|
September 30, 2015
Defective autophagy is a key feature of cerebral cavernous malformations
Saverio Marchi, Mariangela Corricelli, Eliana Trapani, et al.
The Journal of Biological Chemistry
|
January 5, 2002
Integrin-induced epidermal growth factor (EGF) receptor activation requires c-Src and p130Cas and leads to phosphorylation of specific EGF receptor tyrosines
Laura Moro, Laura Dolce, Sara Cabodi, et al.
Circulation
|
December 10, 2014
Strategy for identifying repurposed drugs for the treatment of cerebral cavernous malformation
Christopher C Gibson, Weiquan Zhu, Chadwick T Davis, et al.
Journal of Medical Genetics
|
June 11, 2013
A de novo X;8 translocation creates a PTK2-THOC2 gene fusion with THOC2 expression knockdown in a patient with psychomotor retardation and congenital cerebellar hypoplasia
Eleonora Di Gregorio, Federico T Bianchi, Alfonso Schiavi, et al.
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Showing results (51-60 of 60) with videos related to
Sort By:
Page
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You have reached the last page of results.
This site can display upto 60 results.
Free Radical Biology & Medicine
|
November 25, 2017
KRIT1 loss-of-function induces a chronic Nrf2-mediated adaptive homeostasis that sensitizes cells to oxidative stress: Implication for Cerebral Cavernous Malformation disease
Cinzia Antognelli, Eliana Trapani, Simona Delle Monache, et al.
Scientific Reports
|
August 17, 2017
Up-regulation of NADPH oxidase-mediated redox signaling contributes to the loss of barrier function in KRIT1 deficient endothelium
Luca Goitre, Peter V DiStefano, Andrea Moglia, et al.
Antioxidants (Basel, Switzerland)
|
January 20, 2019
KRIT1 Loss-Of-Function Associated with Cerebral Cavernous Malformation Disease Leads to Enhanced <i>S</i>-Glutathionylation of Distinct Structural and Regulatory Proteins
Laura Cianfruglia, Andrea Perrelli, Claudia Fornelli, et al.
Brain Pathology (Zurich, Switzerland)
|
October 30, 2010
Mutation analysis of CCM1, CCM2 and CCM3 genes in a cohort of Italian patients with cerebral cavernous malformation
Rosalia D'Angelo, Valeria Marini, Carmela Rinaldi, et al.
International Journal of Molecular Sciences
|
October 9, 2019
KRIT1 Deficiency Promotes Aortic Endothelial Dysfunction
Francesco Vieceli Dalla Sega, Raffaella Mastrocola, Giorgio Aquila, et al.
Nature
|
June 11, 2013
EndMT contributes to the onset and progression of cerebral cavernous malformations
Luigi Maddaluno, Noemi Rudini, Roberto Cuttano, et al.
EMBO Molecular Medicine
|
September 30, 2015
Defective autophagy is a key feature of cerebral cavernous malformations
Saverio Marchi, Mariangela Corricelli, Eliana Trapani, et al.
The Journal of Biological Chemistry
|
January 5, 2002
Integrin-induced epidermal growth factor (EGF) receptor activation requires c-Src and p130Cas and leads to phosphorylation of specific EGF receptor tyrosines
Laura Moro, Laura Dolce, Sara Cabodi, et al.
Circulation
|
December 10, 2014
Strategy for identifying repurposed drugs for the treatment of cerebral cavernous malformation
Christopher C Gibson, Weiquan Zhu, Chadwick T Davis, et al.
Journal of Medical Genetics
|
June 11, 2013
A de novo X;8 translocation creates a PTK2-THOC2 gene fusion with THOC2 expression knockdown in a patient with psychomotor retardation and congenital cerebellar hypoplasia
Eleonora Di Gregorio, Federico T Bianchi, Alfonso Schiavi, et al.
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