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Scot C Leary

Showing results (51-60 of 55) with videos related to

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Cell Reports|September 15, 2021
Homeostatic control of nuclear-encoded mitochondrial gene expression by the histone variant H2A.Z is essential for neuronal survivalChristopher Lowden, Aren Boulet, Nicholas A Boehler, et al.
Cell Reports|February 17, 2015
The Mitochondrial Metallochaperone SCO1 Is Required to Sustain Expression of the High-Affinity Copper Transporter CTR1 and Preserve Copper HomeostasisChristopher J Hlynialuk, Binbing Ling, Zakery N Baker, et al.
Biorxiv : the Preprint Server for Biology|June 4, 2026
Mitochondrial copper stabilizes lipoylated TCA cycle proteins to sustain metabolism and proliferationSantanu Ghosh, Andrew F Jarvis, Jordi C J Hintzen, et al.
The Journal of Clinical Investigation|October 27, 2022
Mitochondrial dysfunction reactivates α-fetoprotein expression that drives copper-dependent immunosuppression in mitochondrial disease modelsKimberly A Jett, Zakery N Baker, Amzad Hossain, et al.
Cell Genomics|May 10, 2025
Identification of targetable vulnerabilities of PLK1-overexpressing cancers by synthetic dosage lethalityChelsea E Cunningham, Frederick S Vizeacoumar, Yue Zhang, et al.
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Showing results (51-60 of 55) with videos related to

Sort By:
Pageof 6
You have reached the last page of results.This site can display upto 55 results.
Cell Reports|September 15, 2021
Homeostatic control of nuclear-encoded mitochondrial gene expression by the histone variant H2A.Z is essential for neuronal survivalChristopher Lowden, Aren Boulet, Nicholas A Boehler, et al.
Cell Reports|February 17, 2015
The Mitochondrial Metallochaperone SCO1 Is Required to Sustain Expression of the High-Affinity Copper Transporter CTR1 and Preserve Copper HomeostasisChristopher J Hlynialuk, Binbing Ling, Zakery N Baker, et al.
Biorxiv : the Preprint Server for Biology|June 4, 2026
Mitochondrial copper stabilizes lipoylated TCA cycle proteins to sustain metabolism and proliferationSantanu Ghosh, Andrew F Jarvis, Jordi C J Hintzen, et al.
The Journal of Clinical Investigation|October 27, 2022
Mitochondrial dysfunction reactivates α-fetoprotein expression that drives copper-dependent immunosuppression in mitochondrial disease modelsKimberly A Jett, Zakery N Baker, Amzad Hossain, et al.
Cell Genomics|May 10, 2025
Identification of targetable vulnerabilities of PLK1-overexpressing cancers by synthetic dosage lethalityChelsea E Cunningham, Frederick S Vizeacoumar, Yue Zhang, et al.
Pageof 6