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Scott E Nelson

Showing results (11-20 of 19) with videos related to

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Plos Pathogens|June 26, 2025
IRF6 controls Epstein-Barr virus (EBV) lytic reactivation and differentiation in EBV-infected epithelial cellsStuart A Fogarty, Deo R Singh, Scott E Nelson, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 8, 2023
Epstein-Barr virus LMP1 protein promotes proliferation and inhibits differentiation of epithelial cells via activation of YAP and TAZDeo R Singh, Scott E Nelson, Abigail S Pawelski, et al.
Plos Pathogens|April 15, 2024
Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in miceJillian A Bristol, Scott E Nelson, Makoto Ohashi, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 26, 2026
Epstein-Barr virus (EBV) infection causes human germinal center B cell-derived lymphomas in the absence of EBNA2 expressionChunyan Wang, Jillian A Bristol, Scott E Nelson, et al.
Plos Pathogens|June 17, 2020
EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized miceChunrong Li, James C Romero-Masters, Shane Huebner, et al.
Journal of Virology|March 6, 2020
An EBNA3A-Mutated Epstein-Barr Virus Retains the Capacity for Lymphomagenesis in a Cord Blood-Humanized Mouse ModelJames C Romero-Masters, Makoto Ohashi, Reza Djavadian, et al.
Plos Pathogens|April 26, 2022
Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cellsJillian A Bristol, Joshua Brand, Makoto Ohashi, et al.
Plos Pathogens|October 3, 2022
Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytesDeo R Singh, Scott E Nelson, Abigail S Pawelski, et al.
Plos Pathogens|February 15, 2020
B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infectionJames C Romero-Masters, Shane M Huebner, Makoto Ohashi, et al.
Pageof 2

Showing results (11-20 of 19) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 19 results.
Plos Pathogens|June 26, 2025
IRF6 controls Epstein-Barr virus (EBV) lytic reactivation and differentiation in EBV-infected epithelial cellsStuart A Fogarty, Deo R Singh, Scott E Nelson, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 8, 2023
Epstein-Barr virus LMP1 protein promotes proliferation and inhibits differentiation of epithelial cells via activation of YAP and TAZDeo R Singh, Scott E Nelson, Abigail S Pawelski, et al.
Plos Pathogens|April 15, 2024
Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in miceJillian A Bristol, Scott E Nelson, Makoto Ohashi, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 26, 2026
Epstein-Barr virus (EBV) infection causes human germinal center B cell-derived lymphomas in the absence of EBNA2 expressionChunyan Wang, Jillian A Bristol, Scott E Nelson, et al.
Plos Pathogens|June 17, 2020
EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized miceChunrong Li, James C Romero-Masters, Shane Huebner, et al.
Journal of Virology|March 6, 2020
An EBNA3A-Mutated Epstein-Barr Virus Retains the Capacity for Lymphomagenesis in a Cord Blood-Humanized Mouse ModelJames C Romero-Masters, Makoto Ohashi, Reza Djavadian, et al.
Plos Pathogens|April 26, 2022
Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cellsJillian A Bristol, Joshua Brand, Makoto Ohashi, et al.
Plos Pathogens|October 3, 2022
Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytesDeo R Singh, Scott E Nelson, Abigail S Pawelski, et al.
Plos Pathogens|February 15, 2020
B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infectionJames C Romero-Masters, Shane M Huebner, Makoto Ohashi, et al.
Pageof 2