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International Journal of Molecular Sciences|February 7, 2020
The Diabetic Cardiac Fibroblast: Mechanisms Underlying Phenotype and FunctionScott P Levick, Alexander WidiapradjaInternational Journal of Molecular Sciences|January 13, 2018
Mast Cells: Key Contributors to Cardiac FibrosisScott P Levick, Alexander WidiapradjaCellular and Molecular Life Sciences : CMLS|January 19, 2017
The role of neuropeptides in adverse myocardial remodeling and heart failureAlexander Widiapradja, Prasad Chunduri, Scott P LevickCellular and Molecular Life Sciences : CMLS|December 7, 2024
The orphan nuclear receptor Nr4a1 contributes to interstitial cardiac fibrosis via modulation of cardiac fibroblast and macrophage phenotypeAlexander Widiapradja, Heather Connery, Martyn Bullock, et al.Endocrinology|September 26, 2022
Foxe1 Deletion in the Adult Mouse Is Associated With Increased Thyroidal Mast Cells and HypothyroidismGrace Lim, Alexander Widiapradja, Scott P Levick, et al.International Journal of Molecular Sciences|December 29, 2020
The Histamine 3 Receptor Is Expressed in the Heart and Its Activation Opposes Adverse Cardiac Remodeling in the Angiotensin II Mouse ModelSamuel L McCaffrey, Grace Lim, Martyn Bullock, et al.Biomedicine & Pharmacotherapy = Biomedecine & Pharmacotherapie|February 9, 2023
Replacement substance P reduces cardiac fibrosis in monkeys with type 2 diabetesGiselle C Meléndez, Kylie Kavanagh, Nazli Gharraee, et al.Cells|October 23, 2021
Replacement of Lost Substance P Reduces Fibrosis in the Diabetic Heart by Preventing Adverse Fibroblast and Macrophage Phenotype ChangesAlexander Widiapradja, Ainsley O Kasparian, Samuel L McCaffrey, et al.American Journal of Physiology. Cell Physiology|May 27, 2024
Cellular metabolism of substance P produces neurokinin-1 receptor peptide agonists with diminished cyclic AMP signalingTamas Kriska, Jayashree Natarajan, Anja Herrnreiter, et al.Pageof 6