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Scott R Barnum

Showing results (51-60 of 79) with videos related to

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JCI Insight|October 5, 2016
Soluble membrane attack complex is diagnostic for intraventricular shunt infection in childrenTheresa N Ramos, Anastasia A Arynchyna, Tessa E Blackburn, et al.
The Journal of Biological Chemistry|June 13, 2012
The C5 convertase is not required for activation of the terminal complement pathway in murine experimental cerebral malariaTheresa N Ramos, Meghan M Darley, Sebastian Weckbach, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 8, 2005
Critical requirement of CD11b (Mac-1) on T cells and accessory cells for development of experimental autoimmune encephalomyelitisDaniel C Bullard, Xianzhen Hu, Trenton R Schoeb, et al.
Journal of Neuroscience Research|June 7, 2007
Signaling through C5aR is not involved in basal neurogenesisYalda Rahpeymai Bogestål, Scott R Barnum, Peter L P Smith, et al.
Neuroscience Letters|September 13, 2005
Expression of C5a in the brain does not exacerbate experimental autoimmune encephalomyelitisRachael Reiman, Antonio Campos Torres, Brian K Martin, et al.
Journal of Immunology (Baltimore, Md. : 1950)|January 5, 2007
Intercellular adhesion molecule-1 expression is required on multiple cell types for the development of experimental autoimmune encephalomyelitisDaniel C Bullard, Xianzhen Hu, Trenton R Schoeb, et al.
Molecular Immunology|January 23, 2013
Therapeutic inhibition of the alternative complement pathway attenuates chronic EAEXianzhen Hu, V Michael Holers, Joshua M Thurman, et al.
Plos One|December 14, 2018
Epidemiology of lumbar punctures in hospitalized patients in the United StatesAdrienne Vickers, John P Donnelly, Justin Xavier Moore, et al.
The Journal of Biological Chemistry|March 16, 2013
Experimental cerebral malaria develops independently of endothelial expression of intercellular adhesion molecule-1 (icam-1)Theresa N Ramos, Daniel C Bullard, Meghan M Darley, et al.
Neuroscience Letters|July 19, 2008
Deletion of both ICAM-1 and C3 enhances severity of experimental autoimmune encephalomyelitis compared to C3-deficient miceSherry S Smith, Michael Ludwig, Jillian E Wohler, et al.
Pageof 8

Showing results (51-60 of 79) with videos related to

Sort By:
Pageof 8
JCI Insight|October 5, 2016
Soluble membrane attack complex is diagnostic for intraventricular shunt infection in childrenTheresa N Ramos, Anastasia A Arynchyna, Tessa E Blackburn, et al.
The Journal of Biological Chemistry|June 13, 2012
The C5 convertase is not required for activation of the terminal complement pathway in murine experimental cerebral malariaTheresa N Ramos, Meghan M Darley, Sebastian Weckbach, et al.
Journal of Immunology (Baltimore, Md. : 1950)|November 8, 2005
Critical requirement of CD11b (Mac-1) on T cells and accessory cells for development of experimental autoimmune encephalomyelitisDaniel C Bullard, Xianzhen Hu, Trenton R Schoeb, et al.
Journal of Neuroscience Research|June 7, 2007
Signaling through C5aR is not involved in basal neurogenesisYalda Rahpeymai Bogestål, Scott R Barnum, Peter L P Smith, et al.
Neuroscience Letters|September 13, 2005
Expression of C5a in the brain does not exacerbate experimental autoimmune encephalomyelitisRachael Reiman, Antonio Campos Torres, Brian K Martin, et al.
Journal of Immunology (Baltimore, Md. : 1950)|January 5, 2007
Intercellular adhesion molecule-1 expression is required on multiple cell types for the development of experimental autoimmune encephalomyelitisDaniel C Bullard, Xianzhen Hu, Trenton R Schoeb, et al.
Molecular Immunology|January 23, 2013
Therapeutic inhibition of the alternative complement pathway attenuates chronic EAEXianzhen Hu, V Michael Holers, Joshua M Thurman, et al.
Plos One|December 14, 2018
Epidemiology of lumbar punctures in hospitalized patients in the United StatesAdrienne Vickers, John P Donnelly, Justin Xavier Moore, et al.
The Journal of Biological Chemistry|March 16, 2013
Experimental cerebral malaria develops independently of endothelial expression of intercellular adhesion molecule-1 (icam-1)Theresa N Ramos, Daniel C Bullard, Meghan M Darley, et al.
Neuroscience Letters|July 19, 2008
Deletion of both ICAM-1 and C3 enhances severity of experimental autoimmune encephalomyelitis compared to C3-deficient miceSherry S Smith, Michael Ludwig, Jillian E Wohler, et al.
Pageof 8