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Journal of Immunology (Baltimore, Md. : 1950)|March 2, 2010
MHC class I molecules with Superenhanced CD8 binding properties bypass the requirement for cognate TCR recognition and nonspecifically activate CTLsLinda Wooldridge, Mathew Clement, Anna Lissina, et al.
Immunology and Cell Biology|February 6, 2016
Identification of human viral protein-derived ligands recognized by individual MHCI-restricted T-cell receptorsBarbara Szomolay, Jie Liu, Paul E Brown, et al.
The Journal of Experimental Medicine|June 23, 2010
Allelic polymorphism in the T cell receptor and its impact on immune responsesStephanie Gras, Zhenjun Chen, John J Miles, et al.
Nature Communications|September 3, 2013
IMGT/HighV QUEST paradigm for T cell receptor IMGT clonotype diversity and next generation repertoire immunoprofilingShuo Li, Marie-Paule Lefranc, John J Miles, et al.
Nature Immunology|September 28, 2005
T cell receptor recognition of a 'super-bulged' major histocompatibility complex class I-bound peptideFleur E Tynan, Scott R Burrows, Ashley M Buckle, et al.
Journal of Immunology (Baltimore, Md. : 1950)|June 11, 2013
HLA peptide length preferences control CD8+ T cell responsesMelissa J Rist, Alex Theodossis, Nathan P Croft, et al.
Journal of Immunology (Baltimore, Md. : 1950)|June 17, 2011
Anti-CD8 antibodies can trigger CD8+ T cell effector function in the absence of TCR engagement and improve peptide-MHCI tetramer stainingMathew Clement, Kristin Ladell, Julia Ekeruche-Makinde, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 17, 2010
Antigen-driven patterns of TCR bias are shared across diverse outcomes of human hepatitis C virus infectionJohn J Miles, Duangtawan Thammanichanond, Sarah Moneer, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 21, 2010
Hard wiring of T cell receptor specificity for the major histocompatibility complex is underpinned by TCR adaptabilityScott R Burrows, Zhenjun Chen, Julia K Archbold, et al.
The Journal of Experimental Medicine|August 27, 2003
A naturally selected dimorphism within the HLA-B44 supertype alters class I structure, peptide repertoire, and T cell recognitionWhitney A Macdonald, Anthony W Purcell, Nicole A Mifsud, et al.
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