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Proceedings of the National Academy of Sciences of the United States of America|March 20, 2013
Molecular chaperone Hsp110 rescues a vesicle transport defect produced by an ALS-associated mutant SOD1 protein in squid axoplasmYuyu Song, Maria Nagy, Weiming Ni, et al.
Cells|October 13, 2023
Axon-Autonomous Effects of the Amyloid Precursor Protein Intracellular Domain (AICD) on Kinase Signaling and Fast Axonal TransportSvenja König, Nadine Schmidt, Karin Bechberger, et al.
Biochemistry|March 26, 2008
Conventional kinesin holoenzymes are composed of heavy and light chain homodimersScott R DeBoer, YiMei You, Anita Szodorai, et al.
Magma (New York, N.Y.)|February 17, 2019
Detection of axonal degeneration in a mouse model of Huntington's disease: comparison between diffusion tensor imaging and anomalous diffusion metricsRodolfo G Gatto, Allen Q Ye, Luis Colon-Perez, et al.
Human Molecular Genetics|July 1, 2015
Analysis of YFP(J16)-R6/2 reporter mice and postmortem brains reveals early pathology and increased vulnerability of callosal axons in Huntington's diseaseRodolfo G Gatto, Yaping Chu, Allen Q Ye, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 8, 2011
Pathogenic forms of tau inhibit kinesin-dependent axonal transport through a mechanism involving activation of axonal phosphotransferasesNicholas M Kanaan, Gerardo A Morfini, Nichole E LaPointe, et al.
The Journal of Biological Chemistry|April 30, 2002
Ca2+-dependent dephosphorylation of kinesin heavy chain on beta-granules in pancreatic beta-cells. Implications for regulated beta-granule transport and insulin exocytosisMatthew J Donelan, Gerardo Morfini, Richard Julyan, et al.
Neuron|October 7, 2003
Neuropathogenic forms of huntingtin and androgen receptor inhibit fast axonal transportGyörgyi Szebenyi, Gerardo A Morfini, Alyssa Babcock, et al.
Journal of Parkinson'S Disease|August 7, 2016
Is Axonal Degeneration a Key Early Event in Parkinson's Disease?Zuzanna Kurowska, Jeffrey H Kordower, A Jon Stoessl, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 14, 2013
The sphingolipid psychosine inhibits fast axonal transport in Krabbe disease by activation of GSK3β and deregulation of molecular motorsLudovico Cantuti Castelvetri, Maria I Givogri, Amy Hebert, et al.
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