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Molecular Neurobiology|May 20, 2019
EGF Treatment Improves Motor Behavior and Cortical GABAergic Function in the R6/2 Mouse Model of Huntington's DiseaseFelecia M Marottoli, Mercedes Priego, Eden Flores-Barrera, et al.Scientific Reports|March 10, 2017
ALS-linked FUS exerts a gain of toxic function involving aberrant p38 MAPK activationReddy Ranjith K Sama, Claudia Fallini, Rodolfo Gatto, et al.Nature Neuroscience|June 16, 2009
Pathogenic huntingtin inhibits fast axonal transport by activating JNK3 and phosphorylating kinesinGerardo A Morfini, Yi-Mei You, Sarah L Pollema, et al.Plos One|December 21, 2017
Prion protein inhibits fast axonal transport through a mechanism involving casein kinase 2Emiliano Zamponi, Fiamma Buratti, Gabriel Cataldi, et al.Frontiers in Molecular Neuroscience|May 27, 2022
CRISPR-Cas9 Knock-In of T513M and G41S Mutations in the Murine β-Galactosyl-Ceramidase Gene Re-capitulates Early-Onset and Adult-Onset Forms of Krabbe DiseaseRima Rebiai, Emily Rue, Steve Zaldua, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 5, 2005
Axonal transport, amyloid precursor protein, kinesin-1, and the processing apparatus: revisitedOrly Lazarov, Gerardo A Morfini, Edward B Lee, et al.Brain : a Journal of Neurology|August 26, 2023
Toxic effects of mutant huntingtin in axons are mediated by its proline-rich domainScott T Brady, Nichole A Mesnard-Hoaglin, Sarah Mays, et al.Nature Neuroscience|October 19, 2010
Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALSDaryl A Bosco, Gerardo Morfini, N Murat Karabacak, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 16, 2009
Axonal transport defects in neurodegenerative diseasesGerardo A Morfini, Matthew Burns, Lester I Binder, et al.Plos One|June 19, 2013
Inhibition of fast axonal transport by pathogenic SOD1 involves activation of p38 MAP kinaseGerardo A Morfini, Daryl A Bosco, Hannah Brown, et al.Pageof 7