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Biochimica Et Biophysica Acta. Molecular Basis of Disease|June 21, 2017
Changes in podocyte TRPC channels evoked by plasma and sera from patients with recurrent FSGS and by putative glomerular permeability factorsEun Young Kim, Hila Roshanravan, Stuart E Dryer
Biochemistry and Biophysics Reports|January 22, 2019
TRPC6 inactivation does not affect loss of renal function in nephrotoxic serum glomerulonephritis in rats, but reduces severity of glomerular lesionsEun Young Kim, Parisa Yazdizadeh Shotorbani, Stuart E Dryer
FASEB Bioadvances|March 4, 2020
TRPC6 inactivation does not protect against diabetic kidney disease in streptozotocin (STZ)-treated Sprague-Dawley ratsNaghmeh Hassanzadeh Khayyat, Eun Young Kim, Stuart E Dryer
Biochimica Et Biophysica Acta. Molecular Basis of Disease|October 9, 2018
Mechanisms underlying modulation of podocyte TRPC6 channels by suPAR: Role of NADPH oxidases and Src family tyrosine kinasesEun Young Kim, Naghmeh Hassanzadeh Khayyat, Stuart E Dryer
Physiological Reviews|August 25, 2022
Ion channels and channelopathies in glomeruliAlexander Staruschenko, Rong Ma, Oleg Palygin, et al.
Journal of Neurophysiology|May 28, 2004
A-current expression is regulated by activity but not by target tissues in developing lumbar motoneurons of the chick embryoReema H Casavant, Costa M Colbert, Stuart E Dryer
American Journal of Physiology. Renal Physiology|May 16, 2008
Nephrin binds to the COOH terminus of a large-conductance Ca2+-activated K+ channel isoform and regulates its expression on the cell surfaceEun Young Kim, Kyoung-Jae Choi, Stuart E Dryer
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