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Journal of Neuroscience Research|August 3, 2007
Antidepressant drugs reverse the loss of adult neural stem cells following chronic stressSeiji Hitoshi, Noriko Maruta, Mikito Higashi, et al.
Neurochemical Research|October 30, 2017
Minocycline Directly Enhances the Self-Renewal of Adult Neural Precursor CellsAnri Kuroda, Takahiro Fuchigami, Satoshi Fuke, et al.
The Journal of Physiological Sciences : JPS|January 22, 2025
Depletion of transit amplifying cells in the adult brain does not affect quiescent neural stem cell pool sizeZakiyyah Munirah Mohd Zaki, Anri Kuroda, Naoko Morimura, et al.
The Journal of Physiological Sciences : JPS|September 13, 2023
Depletion of transit amplifying cells in the adult brain does not affect quiescent neural stem cell pool sizeZakiyyah Munirah Mohd Zaki, Anri Kuroda, Naoko Morimura, et al.
Journal of Neurochemistry|October 27, 2006
Regulation of glial development by cystatin CAkiko Hasegawa, Masae Naruse, Seiji Hitoshi, et al.
Frontiers in Neuroscience|February 18, 2020
Early Maternal and Social Deprivation Expands Neural Stem Cell Population Size and Reduces Hippocampus/Amygdala-Dependent Fear MemoryKenny Anak Daun, Takahiro Fuchigami, Natsu Koyama, et al.
Developmental Neuroscience|March 2, 2006
Notch signaling is required to maintain all neural stem cell populations--irrespective of spatial or temporal nicheTania O Alexson, Seiji Hitoshi, Brenda L Coles, et al.
The Journal of Physiological Sciences : JPS|November 19, 2015
Neural stem cells and neuro/gliogenesis in the central nervous system: understanding the structural and functional plasticity of the developing, mature, and diseased brainMasahiro Yamaguchi, Tatsunori Seki, Itaru Imayoshi, et al.
Glycoconjugate Journal|November 24, 2014
Determination of major sialylated N-glycans and identification of branched sialylated N-glycans that dynamically change their content during development in the mouse cerebral cortexTomohiro Torii, Takeshi Yoshimura, Mai Narumi, et al.
Communications Biology|June 24, 2021
Precise CAG repeat contraction in a Huntington's Disease mouse model is enabled by gene editing with SpCas9-NGSeiya Oura, Taichi Noda, Naoko Morimura, et al.
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