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Neurochemical Research|June 26, 2022
Fut9 Deficiency Causes Abnormal Neural Development in the Mouse Cerebral Cortex and RetinaAsmaa Abdullah, Yoshitaka Hayashi, Naoko Morimura, et al.Cerebral Cortex (New York, N.Y. : 1991)|August 9, 2020
Life-Long Neural Stem Cells Are Fate-Specified at an Early Developmental StageAoi Tanaka, Shohei Ishida, Takahiro Fuchigami, et al.Journal of Neuroscience Research|November 23, 2012
Olig2-lineage cells preferentially differentiate into oligodendrocytes but their processes degenerate at the chronic demyelinating stage of proteolipid protein-overexpressing mouseTakahiro Shimizu, Kenji F Tanaka, Hirohide Takebayashi, et al.The Journal of Biological Chemistry|August 30, 2013
The Lewis X-related α1,3-fucosyltransferase, Fut10, is required for the maintenance of stem cell populationsAkhilesh Kumar, Tomohiro Torii, Yugo Ishino, et al.Cerebral Cortex (New York, N.Y. : 1991)|June 26, 2015
The Dorsoventral Boundary of the Germinal Zone is a Specialized Niche for the Generation of Cortical Oligodendrocytes during a Restricted Temporal WindowMasae Naruse, Yugo Ishino, Akhilesh Kumar, et al.Genes & Development|August 4, 2004
Primitive neural stem cells from the mammalian epiblast differentiate to definitive neural stem cells under the control of Notch signalingSeiji Hitoshi, Raewyn M Seaberg, Cheryl Koscik, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 24, 2006
Vascular endothelial growth factor directly inhibits primitive neural stem cell survival but promotes definitive neural stem cell survivalTamaki Wada, Jody J Haigh, Masatsugu Ema, et al.Glycobiology|December 19, 2006
Developmental changes in the expression of glycogenes and the content of N-glycans in the mouse cerebral cortexAkihiro Ishii, Takeshi Ikeda, Seiji Hitoshi, et al.Nature Neuroscience|July 19, 2011
Mammalian Gcm genes induce Hes5 expression by active DNA demethylation and induce neural stem cellsSeiji Hitoshi, Yugo Ishino, Akhilesh Kumar, et al.Annals of Clinical and Translational Neurology|December 26, 2014
Heterozygous Polg mutation causes motor dysfunction due to mtDNA deletionsSatoshi Fuke, Mizue Kametani, Kazuyuki Yamada, et al.Pageof 5