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Biorxiv : the Preprint Server for Biology|October 4, 2023
RGS2 is an innate immune checkpoint for TLR4 and Gαq-mediated IFNγ generation and lung injuryJagdish Chandra Joshi, Bhagwati Joshi, Cuiping Zhang, et al.Iscience|January 1, 2024
Erratum: Piezo1 induces endothelial responses to shear stress via soluble adenylyl Cyclase-IP3R2 circuitDianicha Santana Nunez, Asrar B Malik, Quinn Lee, et al.Experimental Eye Research|April 8, 2014
Correspondence of retinal thinning and vasculopathy in mice with oxygen-induced retinopathyOlachi J Mezu-Ndubuisi, Justin Wanek, Felix Y Chau, et al.Kidney International|October 4, 2013
The Nrf2 triterpenoid activator, CDDO-imidazolide, protects kidneys from ischemia-reperfusion injury in miceManchang Liu, Narsa M Reddy, Elizabeth M Higbee, et al.The American Journal of Pathology|January 24, 2012
Targeted deletion of Jun/AP-1 in alveolar epithelial cells causes progressive emphysema and worsens cigarette smoke-induced lung inflammationNarsa M Reddy, Suryanaraya Vegiraju, Ashley Irving, et al.Investigative Ophthalmology & Visual Science|September 21, 2013
In vivo retinal vascular oxygen tension imaging and fluorescein angiography in the mouse model of oxygen-induced retinopathyOlachi J Mezu-Ndubuisi, Pang-yu Teng, Justin Wanek, et al.The Journal of Cell Biology|October 15, 2021
Tyrosine phosphorylation of S1PR1 leads to chaperone BiP-mediated import to the endoplasmic reticulumMumtaz Anwar, Md Ruhul Amin, Vijay Avin Balaji Ragunathrao, et al.American Journal of Physiology. Lung Cellular and Molecular Physiology|June 18, 2013
Conditional deletion of FAK in mice endothelium disrupts lung vascular barrier function due to destabilization of RhoA and Rac1 activitiesTracy Thennes Schmidt, Mohammad Tauseef, Lili Yue, et al.Circulation Research|May 26, 2007
RhoGDI-1 modulation of the activity of monomeric RhoGTPase RhoA regulates endothelial barrier function in mouse lungsMatvey Gorovoy, Radu Neamu, Jiaxin Niu, et al.American Journal of Physiology. Cell Physiology|February 13, 2015
Upregulated expression of STIM2, TRPC6, and Orai2 contributes to the transition of pulmonary arterial smooth muscle cells from a contractile to proliferative phenotypeRuby A Fernandez, Jun Wan, Shanshan Song, et al.Pageof 19