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Current Diabetes Reports|October 6, 2017
Gene Editing and Human Pluripotent Stem Cells: Tools for Advancing Diabetes Disease Modeling and Beta-Cell DevelopmentKatelyn Millette, Senta GeorgiaDiabetes|October 27, 2006
p27 Regulates the transition of beta-cells from quiescence to proliferationSenta Georgia, Anil BhushanThe Journal of Clinical Investigation|October 7, 2004
Beta cell replication is the primary mechanism for maintaining postnatal beta cell massSenta Georgia, Anil BhushanFrontiers in Endocrinology|April 19, 2021
The Effects of Non-Nutritive Sweetener Consumption in the Pediatric Populations: What We Know, What We Don't, and What We Need to LearnBetty Shum, Senta GeorgiaCurrent Opinion in Cell Biology|December 7, 2007
Formation and regeneration of the endocrine pancreasSangeeta Dhawan, Senta Georgia, Anil BhushanGenes & Development|February 23, 2013
DNMT1 represses p53 to maintain progenitor cell survival during pancreatic organogenesisSenta Georgia, Murtaza Kanji, Anil BhushanDiabetes Care|April 27, 2021
Spike in Diabetic Ketoacidosis Rates in Pediatric Type 2 Diabetes During the COVID-19 PandemicLily C Chao, Alaina P Vidmar, Senta GeorgiaMolecular Endocrinology (Baltimore, Md.)|October 8, 2011
Skp2 is required for incretin hormone-mediated β-cell proliferationShuen-Ing Tschen, Senta Georgia, Sangeeta Dhawan, et al.American Journal of Physiology. Endocrinology and Metabolism|July 1, 2010
Inconsistent formation and nonfunction of insulin-positive cells from pancreatic endoderm derived from human embryonic stem cells in athymic nude ratsAleksey V Matveyenko, Senta Georgia, Anil Bhushan, et al.Developmental Biology|August 11, 2006
p57 and Hes1 coordinate cell cycle exit with self-renewal of pancreatic progenitorsSenta Georgia, Rosemary Soliz, Min Li, et al.Pageof 3