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Seokjin Ham

Showing results (11-20 of 34) with videos related to

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Aging Cell|March 26, 2024
Combinatorial transcriptomic and genetic dissection of insulin/IGF-1 signaling-regulated longevity in Caenorhabditis elegansSeokjin Ham, Sieun S Kim, Sangsoon Park, et al.
Genome Research|November 9, 2022
Systematic transcriptome analysis associated with physiological and chronological aging in <i>Caenorhabditis elegans</i>Seokjin Ham, Sieun S Kim, Sangsoon Park, et al.
Aging Cell|August 15, 2025
T-CLASS: An Online Tool for the Identification and Classification of Aging and Senescence Using Transcriptome DataSeung-Chul J Lee, Gee-Yoon Lee, Sieun S Kim, et al.
The Journal of Cell Biology|March 5, 2021
Reduced insulin/IGF1 signaling prevents immune aging via ZIP-10/bZIP-mediated feedforward loopYujin Lee, Yoonji Jung, Dae-Eun Jeong, et al.
Nature Communications|September 21, 2019
Transcription-dependent targeting of Hda1C to hyperactive genes mediates H4-specific deacetylation in yeastSo Dam Ha, Seokjin Ham, Min Young Kim, et al.
Plos One|February 3, 2017
Identification of the early and late responder genes during the generation of induced pluripotent stem cells from mouse fibroblastsJihwan Park, Yoo-Wook Kwon, Seokjin Ham, et al.
Plos Genetics|March 5, 2020
Inhibition of the oligosaccharyl transferase in Caenorhabditis elegans that compromises ER proteostasis suppresses p38-dependent protection against pathogenic bacteriaDae-Eun Jeong, Yujin Lee, Seokjin Ham, et al.
Nature Communications|September 25, 2021
A PTEN variant uncouples longevity from impaired fitness in Caenorhabditis elegans with reduced insulin/IGF-1 signalingHae-Eun H Park, Wooseon Hwang, Seokjin Ham, et al.
Scientific Reports|May 6, 2022
Dietary antigens suppress the proliferation of type 2 innate lymphoid cells by restraining homeostatic IL-25 productionMinji Lee, Hyun-Ja Ko, Sung-Wook Hong, et al.
Autophagy|July 4, 2024
HLH-30/TFEB mediates sexual dimorphism in immunity in <i>Caenorhabditis elegans</i>Jooyeon Sohn, Sujeong Kwon, Gee-Yoon Lee, et al.
Pageof 4

Showing results (11-20 of 34) with videos related to

Sort By:
Pageof 4
Aging Cell|March 26, 2024
Combinatorial transcriptomic and genetic dissection of insulin/IGF-1 signaling-regulated longevity in Caenorhabditis elegansSeokjin Ham, Sieun S Kim, Sangsoon Park, et al.
Genome Research|November 9, 2022
Systematic transcriptome analysis associated with physiological and chronological aging in <i>Caenorhabditis elegans</i>Seokjin Ham, Sieun S Kim, Sangsoon Park, et al.
Aging Cell|August 15, 2025
T-CLASS: An Online Tool for the Identification and Classification of Aging and Senescence Using Transcriptome DataSeung-Chul J Lee, Gee-Yoon Lee, Sieun S Kim, et al.
The Journal of Cell Biology|March 5, 2021
Reduced insulin/IGF1 signaling prevents immune aging via ZIP-10/bZIP-mediated feedforward loopYujin Lee, Yoonji Jung, Dae-Eun Jeong, et al.
Nature Communications|September 21, 2019
Transcription-dependent targeting of Hda1C to hyperactive genes mediates H4-specific deacetylation in yeastSo Dam Ha, Seokjin Ham, Min Young Kim, et al.
Plos One|February 3, 2017
Identification of the early and late responder genes during the generation of induced pluripotent stem cells from mouse fibroblastsJihwan Park, Yoo-Wook Kwon, Seokjin Ham, et al.
Plos Genetics|March 5, 2020
Inhibition of the oligosaccharyl transferase in Caenorhabditis elegans that compromises ER proteostasis suppresses p38-dependent protection against pathogenic bacteriaDae-Eun Jeong, Yujin Lee, Seokjin Ham, et al.
Nature Communications|September 25, 2021
A PTEN variant uncouples longevity from impaired fitness in Caenorhabditis elegans with reduced insulin/IGF-1 signalingHae-Eun H Park, Wooseon Hwang, Seokjin Ham, et al.
Scientific Reports|May 6, 2022
Dietary antigens suppress the proliferation of type 2 innate lymphoid cells by restraining homeostatic IL-25 productionMinji Lee, Hyun-Ja Ko, Sung-Wook Hong, et al.
Autophagy|July 4, 2024
HLH-30/TFEB mediates sexual dimorphism in immunity in <i>Caenorhabditis elegans</i>Jooyeon Sohn, Sujeong Kwon, Gee-Yoon Lee, et al.
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