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Molecular Biosystems|February 21, 2015
Differential proteome-metabolome profiling of YCA1-knock-out and wild type cells reveals novel metabolic pathways and cellular processes dependent on the yeast metacaspaseMaša Ždralević, Valentina Longo, Nicoletta Guaragnella, et al.
Frontiers in Oncology|December 19, 2017
Mitochondrial Dysfunction: A Novel Potential Driver of Epithelial-to-Mesenchymal Transition in CancerFlora Guerra, Nicoletta Guaragnella, Arnaldo A Arbini, et al.
Journal of Proteomics|August 14, 2015
Proteome and metabolome profiling of wild-type and YCA1-knock-out yeast cells during acetic acid-induced programmed cell deathValentina Longo, Maša Ždralević, Nicoletta Guaragnella, et al.
FEBS Letters|April 9, 2008
Cytochrome c is released from coupled mitochondria of yeast en route to acetic acid-induced programmed cell death and can work as an electron donor and a ROS scavengerSergio Giannattasio, Anna Atlante, Lucia Antonacci, et al.
International Journal of Molecular Sciences|March 28, 2026
Enhanced Untargeted Metabolomics Based on High-Resolution Mass Spectrometry Reveals Global Rewiring Due to Mitochondrial Dysfunction in YeastFabrizio Mastrorocco, Luca De Martino, Igor Fochi, et al.
Biochimica Et Biophysica Acta|June 14, 2005
An increase in the ATP levels occurs in cerebellar granule cells en route to apoptosis in which ATP derives from both oxidative phosphorylation and anaerobic glycolysisAnna Atlante, Sergio Giannattasio, Antonella Bobba, et al.
FEBS Letters|December 13, 2006
YCA1 participates in the acetic acid induced yeast programmed cell death also in a manner unrelated to its caspase-like activityNicoletta Guaragnella, Clara Pereira, Maria João Sousa, et al.
Microorganisms|September 28, 2021
<i>RTG</i> Signaling Sustains Mitochondrial Respiratory Capacity in <i>HOG1</i>-Dependent OsmoadaptationNicoletta Guaragnella, Gennaro Agrimi, Pasquale Scarcia, et al.
Biochimica Et Biophysica Acta|August 3, 2013
Yeast growth in raffinose results in resistance to acetic-acid induced programmed cell death mostly due to the activation of the mitochondrial retrograde pathwayNicoletta Guaragnella, Maša Ždralević, Paolo Lattanzio, et al.
Pathology, Research and Practice|August 10, 2024
miR-29a expression negatively correlates with Bcl-2 levels in colorectal cancer and is correlated with better prognosisJanja Raonić, Maša Ždralević, Ljiljana Vučković, et al.
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