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Oxidative Medicine and Cellular Longevity|November 18, 2015
Tau Hyperphosphorylation and Oxidative Stress, a Critical Vicious Circle in Neurodegenerative Tauopathies?Seyedeh Maryam Alavi Naini, Nadia Soussi-YanicostasFrontiers in Cell and Developmental Biology|January 9, 2019
Heparan Sulfate as a Therapeutic Target in Tauopathies: Insights From ZebrafishSeyedeh Maryam Alavi Naini, Nadia Soussi-YanicostasMolecules (Basel, Switzerland)|October 27, 2018
Vipers of the Middle East: A Rich Source of Bioactive MoleculesMohamad Rima, Seyedeh Maryam Alavi Naini, Marc Karam, et al.Translational Neurodegeneration|March 24, 2018
Surfen and oxalyl surfen decrease tau hyperphosphorylation and mitigate neuron deficits in vivo in a zebrafish model of tauopathySeyedeh Maryam Alavi Naini, Constantin Yanicostas, Rahma Hassan-Abdi, et al.Neurobiology of Disease|July 18, 2012
Spatacsin and spastizin act in the same pathway required for proper spinal motor neuron axon outgrowth in zebrafishElodie Martin, Constantin Yanicostas, Agnès Rastetter, et al.Acta Neuropathologica Communications|December 21, 2017
What is the evidence that tau pathology spreads through prion-like propagation?Amrit Mudher, Morvane Colin, Simon Dujardin, et al.Current Biology : CB|September 16, 2021
Trans-inhibition of axon terminals underlies competition in the habenulo-interpeduncular pathwayMargherita Zaupa, Seyedeh Maryam Alavi Naini, Maroun Abi Younes, et al.Human Molecular Genetics|March 8, 2013
ZEB2 zinc-finger missense mutations lead to hypomorphic alleles and a mild Mowat-Wilson syndromeJamal Ghoumid, Loïc Drevillon, Seyedeh Maryam Alavi-Naini, et al.Brain : a Journal of Neurology|April 6, 2015
HS3ST2 expression is critical for the abnormal phosphorylation of tau in Alzheimer's disease-related tau pathologyJulia Elisa Sepulveda-Diaz, Seyedeh Maryam Alavi Naini, Minh Bao Huynh, et al.Pageof 1