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Proceedings of the National Academy of Sciences of the United States of America|July 12, 2013
Pathological hypertrophy amelioration by PRAS40-mediated inhibition of mTORC1Mirko Völkers, Haruhiro Toko, Shirin Doroudgar, et al.
Circulation Research|May 9, 2013
Fibronectin is essential for reparative cardiac progenitor cell response after myocardial infarctionMathias H Konstandin, Haruhiro Toko, Grady M Gastelum, et al.
The Journal of Biological Chemistry|December 31, 2013
Differential regulation of cellular senescence and differentiation by prolyl isomerase Pin1 in cardiac progenitor cellsHaruhiro Toko, Nirmala Hariharan, Mathias H Konstandin, et al.
European Heart Journal|January 14, 2011
Mitochondrial translocation of Nur77 mediates cardiomyocyte apoptosisZhaokang Cheng, Mirko Völkers, Shabana Din, et al.
Physiological Reviews|July 12, 2011
Myocardial AKT: the omnipresent nexusMark A Sussman, Mirko Völkers, Kimberlee Fischer, et al.
Circulation Research|June 12, 2014
Metabolic dysfunction consistent with premature aging results from deletion of Pim kinasesShabana Din, Mathias H Konstandin, Bevan Johnson, et al.
The Journal of Biological Chemistry|April 18, 2015
Functional Effect of Pim1 Depends upon Intracellular Localization in Human Cardiac Progenitor CellsKaitlen Samse, Jacqueline Emathinger, Nirmala Hariharan, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 12, 2008
Pim-1 kinase antagonizes aspects of myocardial hypertrophy and compensation to pathological pressure overloadJohn A Muraski, Kimberlee M Fischer, Weitao Wu, et al.
Circulation Research|March 15, 2013
Regulation of cardiac hypertrophic signaling by prolyl isomerase Pin1Haruhiro Toko, Mathias H Konstandin, Shirin Doroudgar, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 30, 2011
Nucleolar stress is an early response to myocardial damage involving nucleolar proteins nucleostemin and nucleophosminDaniele Avitabile, Brandi Bailey, Christopher T Cottage, et al.
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