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Plos One|December 30, 2021
HPV16 E1 dysregulated cellular genes involved in cell proliferation and host DNA damage: A possible role in cervical carcinogenesisFern Baedyananda, Arkom Chaiwongkot, Shankar Varadarajan, et al.British Journal of Cancer|March 9, 2016
BH3 profiling and a toolkit of BH3-mimetic drugs predict anti-apoptotic dependence of cancer cellsMichael Butterworth, Andrew Pettitt, Shankar Varadarajan, et al.Cell Death & Disease|July 10, 2019
Apogossypol-mediated reorganisation of the endoplasmic reticulum antagonises mitochondrial fission and apoptosisGovindaraju Yedida, Mateus Milani, Gerald M Cohen, et al.Frontiers in Cellular and Infection Microbiology|August 15, 2022
Role of HPV16 E1 in cervical carcinogenesisFern Baedyananda, Thanayod Sasivimolrattana, Arkom Chaiwongkot, et al.Neoplasia (New York, N.Y.)|May 2, 2013
Sabutoclax (BI97C1) and BI112D1, putative inhibitors of MCL-1, induce mitochondrial fragmentation either upstream of or independent of apoptosisShankar Varadarajan, Michael Butterworth, Jun Wei, et al.Cancer Research|April 27, 2021
STINGing Viral Tumors: What We Know from Head and Neck CancersEmma Saulters, John F Woolley, Shankar Varadarajan, et al.Molecular Biology of the Cell|February 8, 2013
The small molecule dispergo tubulates the endoplasmic reticulum and inhibits exportLei Lu, Rami N Hannoush, Brian C Goess, et al.Oncotarget|June 11, 2015
Maritoclax and dinaciclib inhibit MCL-1 activity and induce apoptosis in both a MCL-1-dependent and -independent mannerShankar Varadarajan, Paramasivan Poornima, Mateus Milani, et al.Biological Chemistry|June 21, 2018
Selective BH3-mimetics targeting BCL-2, BCL-XL or MCL-1 induce severe mitochondrial perturbationsKristina Henz, Aoula Al-Zebeeby, Marion Basoglu, et al.Cell Death & Disease|January 13, 2017
DRP-1 is required for BH3 mimetic-mediated mitochondrial fragmentation and apoptosisMateus Milani, Dominic P Byrne, Georgia Greaves, et al.Pageof 2